Petit G, Spitalier-Kaveh H
Ann Parasitol Hum Comp. 1979 Jan-Feb;54(1):81-92.
Histological and ultrastructural study of the adipose tissue of Aedes aegypti has shown that infection with the filaria Dipetalonema dessetae locally induces a highly active synthesis of proteins (formation of ribosomes, endoplasmic reticulum and Golgi complexes, multiplication of mitochondria). This phenomenon appears similar to the effect of beta-ecdysone after the up-take of blood. Mosquitoes let to starve during the development of the filaria show a quick disappearance of storage substances (lipids and glycogen) and involution forms of the induced ergastoplasma (after seventh days only), so that the infected tissue degenerates and the development of the filaria is arrested.
埃及伊蚊脂肪组织的组织学和超微结构研究表明,感染丝状线虫德氏双瓣线虫会在局部诱导蛋白质的高活性合成(核糖体、内质网和高尔基体复合物的形成,线粒体的增殖)。这种现象似乎类似于摄取血液后β-蜕皮激素的作用。在丝状线虫发育过程中挨饿的蚊子,其储存物质(脂质和糖原)迅速消失,诱导的内质网也出现退化形式(仅在第七天后),从而使受感染组织退化,丝状线虫的发育停止。