Dykes J R, Saxton C, Taylor S H
Br Med J. 1969 May 24;2(5655):490. doi: 10.1136/bmj.2.5655.490.
A nearly complete suppression of insulin secretion was observed in four patients in circulatory shock following acute myocardial infarction. This may be due to intense sympathetic activity and raised catecholamine excretion associated with shock.
在急性心肌梗死后发生循环休克的4例患者中,观察到胰岛素分泌几乎完全受到抑制。这可能是由于与休克相关的强烈交感神经活动和儿茶酚胺排泄增加所致。