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The ultrastructure of senile plaques.

作者信息

Luse S A, Smith K R

出版信息

Am J Pathol. 1964 Apr;44(4):553-63.

PMID:5877505
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1907027/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/b154bd5a12c1/amjpathol00305-0045-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/ae5023acd777/amjpathol00305-0041-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/65a277c7284f/amjpathol00305-0041-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/25e5baa9b5af/amjpathol00305-0041-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/4e078c447dc1/amjpathol00305-0042-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/15d9b68098ff/amjpathol00305-0043-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/1dcc70ac773d/amjpathol00305-0044-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/e3c12ccb47d1/amjpathol00305-0044-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/9ceac278bb76/amjpathol00305-0045-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/b154bd5a12c1/amjpathol00305-0045-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/ae5023acd777/amjpathol00305-0041-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/65a277c7284f/amjpathol00305-0041-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/25e5baa9b5af/amjpathol00305-0041-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/4e078c447dc1/amjpathol00305-0042-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/15d9b68098ff/amjpathol00305-0043-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/1dcc70ac773d/amjpathol00305-0044-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/e3c12ccb47d1/amjpathol00305-0044-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/9ceac278bb76/amjpathol00305-0045-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2820/1907027/b154bd5a12c1/amjpathol00305-0045-b.jpg

相似文献

1
The ultrastructure of senile plaques.老年斑的超微结构
Am J Pathol. 1964 Apr;44(4):553-63.
2
[Senile plaques, neuro-fibrillar degeneration and other associated cerebral lesions (morphology and pathogenetic theory].
Schweiz Arch Neurol Neurochir Psychiatr. 1979;124(2):317-33.
3
The contribution of altered synapses in the senile plaque: an electron microscopic study in Alzheimer's dementia.老年斑中突触改变的作用：阿尔茨海默病性痴呆的电子显微镜研究
J Neuropathol Exp Neurol. 1967 Jan;26(1):25-39. doi: 10.1097/00005072-196701000-00003.
4
[Development of senile plaques and neurofibrillary lesions in the cerebral cortex of elderly persons. Statistical study of several anatomo-histoclinical correlates].[老年人大脑皮质中淀粉样斑块和神经原纤维病变的发展。几种解剖组织临床相关性的统计学研究]
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Neuritic (senile) plaques and filamentous changes in aged rhesus monkeys.老年恒河猴的神经炎（老年）斑块和丝状变化。
J Neuropathol Exp Neurol. 1973 Oct;32(4):566-84. doi: 10.1097/00005072-197310000-00007.
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[Ultrastructural study of Pick's disease. Apropos of 3 cases].[匹克氏病的超微结构研究。附3例报告]
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7
[Relation between the degree of severity of the dementia and the extent of the manifestation of the pathomorphological changes in the cerebral cortex in senile dementia and Alzheimer's disease].[老年痴呆症和阿尔茨海默病中痴呆严重程度与大脑皮质病理形态学变化表现程度之间的关系]
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8
Neocortical synapses in a presenile dementia.早老性痴呆中的新皮质突触
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Presenile dementia: some pathological problems and possibilites.早老性痴呆：一些病理学问题及可能性
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10
Isolation and preliminary characterization of Alzheimer plaques from presenile and senile dementia.早老性和老年性痴呆中阿尔茨海默斑的分离与初步特征分析
Trans Am Neurol Assoc. 1970;95:47-50.

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Free Neuropathol. 2022 Jul 8;3:17. doi: 10.17879/freeneuropathology-2022-3806. eCollection 2022 Jan.
2
Natural Bioactive Products and Alzheimer's Disease Pathology: Lessons from Transgenic Models.天然生物活性产物与阿尔茨海默病病理学：转基因模型的启示
Diseases. 2022 May 13;10(2):28. doi: 10.3390/diseases10020028.
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The Causal Role of Lipoxidative Damage in Mitochondrial Bioenergetic Dysfunction Linked to Alzheimer's Disease Pathology.

本文引用的文献

1
[The fine structure of the spleen and liver in experimental amyloidosis].[实验性淀粉样变性中脾脏和肝脏的精细结构]
Z Zellforsch Mikrosk Anat. 1960;52:653-73.
2
Senile cerebral disease: a critical survey of traditional concepts based upon observations with newer technics.老年脑疾病：基于新技术观察对传统概念的批判性审视。
Lab Invest. 1959 Mar-Apr;8(2):335-70.
3
Alzheimer's disease; a clinico-pathologic analysis of twenty-three cases with a theory on pathogenesis.阿尔茨海默病；23例临床病理分析及发病机制理论
脂氧化损伤在与阿尔茨海默病病理相关的线粒体生物能量功能障碍中的因果作用。
Life (Basel). 2021 Apr 25;11(5):388. doi: 10.3390/life11050388.
4
Early Dendritic Dystrophy in Human Brains With Primary Age-Related Tauopathy.患有原发性年龄相关性tau蛋白病的人类大脑中的早期树突状营养不良
Front Aging Neurosci. 2020 Dec 7;12:596894. doi: 10.3389/fnagi.2020.596894. eCollection 2020.
5
Innate Immune and Fungal Model of Alzheimer's Disease.阿尔茨海默病的固有免疫与真菌模型
J Alzheimers Dis Rep. 2018 Aug 1;2(1):139-152. doi: 10.3233/ADR-180073.
6
Sortilin Fragments Deposit at Senile Plaques in Human Cerebrum.sortilin片段沉积于人类大脑的老年斑中。
Front Neuroanat. 2017 Jun 7;11:45. doi: 10.3389/fnana.2017.00045. eCollection 2017.
7
Use of Caenorhabditis elegans as a model to study Alzheimer's disease and other neurodegenerative diseases.利用秀丽隐杆线虫作为研究阿尔茨海默病和其他神经退行性疾病的模型。
Front Genet. 2014 Sep 5;5:279. doi: 10.3389/fgene.2014.00279. eCollection 2014.
8
APL-1, the Alzheimer's Amyloid precursor protein in Caenorhabditis elegans, modulates multiple metabolic pathways throughout development.在秀丽隐杆线虫中，APL-1（阿尔茨海默病淀粉样前体蛋白）在发育过程中调节多种代谢途径。
Genetics. 2012 Jun;191(2):493-507. doi: 10.1534/genetics.112.138768. Epub 2012 Mar 30.
9
Amyloid generation and dysfunctional immunoproteasome activation with disease progression in animal model of familial Alzheimer's disease.家族性阿尔茨海默病动物模型中随着疾病进展的淀粉样生成和免疫蛋白酶体功能障碍激活。
Brain Pathol. 2012 Sep;22(5):636-53. doi: 10.1111/j.1750-3639.2011.00560.x. Epub 2012 Jan 13.
10
Caenorhabditis elegans as a model organism to study APP function.秀丽隐杆线虫作为一种研究 APP 功能的模式生物。
Exp Brain Res. 2012 Apr;217(3-4):397-411. doi: 10.1007/s00221-011-2905-7. Epub 2011 Oct 29.
J Nerv Ment Dis. 1953 Aug;118(2):97-130.