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1
Synthesis of clotting factors by the isolated perfused rat liver.离体灌注大鼠肝脏合成凝血因子。
J Clin Invest. 1966 May;45(5):690-701. doi: 10.1172/JCI105384.
2
Release of vitamin K-dependent coagulation factors by isolated perfused rat liver.
Am J Physiol. 1968 Apr;214(4):919-22. doi: 10.1152/ajplegacy.1968.214.4.919.
3
Synthesis of clotting factors by a cell-free system from rat liver in response to the addition of vitamin K1 in vitro.大鼠肝脏无细胞体系在体外添加维生素K1后凝血因子的合成。
Biochem Biophys Res Commun. 1977 Jan 10;74(1):25-32. doi: 10.1016/0006-291x(77)91370-5.
4
Vitamin K and the synthesis of factors VII-X by isolated rat liver cells.维生素K与离体大鼠肝细胞合成因子VII - X的关系。
Proc Soc Exp Biol Med. 1969 Nov;132(2):509-13. doi: 10.3181/00379727-132-34248.
5
Reversal by vitamin K of cycloheximide inhibited biosynthesis of prothrombin in the isolated perfused rat liver.维生素K可逆转环己酰亚胺对离体灌注大鼠肝脏中凝血酶原生物合成的抑制作用。
Biochem Biophys Res Commun. 1970 Mar 27;38(6):1041-8. doi: 10.1016/0006-291x(70)90344-x.
6
[Role of liver maintenance of normal hemostasis].[肝脏维持正常止血功能的作用]
Pol Tyg Lek. 1974 Jun 3;30(22):949-52.
7
The mode of action of vitamin K in stimulating prothrombin synthesis.
Thromb Diath Haemorrh. 1968 Jul 31;19(3):611-2.
8
Vitamin K and warfarin. Metabolism, function and interaction.维生素K与华法林：代谢、功能及相互作用
Am J Med. 1972 Sep;53(3):261-7. doi: 10.1016/0002-9343(72)90167-2.
9
Failure of actinomycin D to inhibit appearance of clotting activity by vitamin K in vitro.放线菌素D在体外不能抑制维生素K诱导的凝血活性的出现。
Experientia. 1967 Jun 15;23(6):421-2. doi: 10.1007/BF02142151.
10
Vitamin K--dependent clotting factors.
Semin Thromb Hemost. 1977 Summer;4(1):1-14.

引用本文的文献

1
Neutrophil-derived migrasomes are an essential part of the coagulation system.中性粒细胞衍生的迁移小体是凝血系统的重要组成部分。
Nat Cell Biol. 2024 Jul;26(7):1110-1123. doi: 10.1038/s41556-024-01440-9. Epub 2024 Jul 12.
2
The Utilisation of INR to identify coagulopathy in burn patients.INR 在烧伤患者凝血功能障碍中的应用。
PLoS One. 2024 Feb 23;19(2):e0278658. doi: 10.1371/journal.pone.0278658. eCollection 2024.
3
"iPSC-derived liver organoids and inherited bleeding disorders: Potential and future perspectives".诱导多能干细胞衍生的肝类器官与遗传性出血性疾病：潜力与未来展望
Front Physiol. 2023 Jan 13;14:1094249. doi: 10.3389/fphys.2023.1094249. eCollection 2023.
4
Post-translational carboxylation of preprothrombin.凝血酶原前体的翻译后羧化作用。
Mol Cell Biochem. 1981 Aug 11;38 Spec No(Pt 1):77-121. doi: 10.1007/BF00235690.
5
Immunohistochemical identification of factor X-like antigen in the A cells of the normal human pancreas.正常人胰腺A细胞中X因子样抗原的免疫组织化学鉴定
Diabetologia. 1982 Sep;23(3):255-60. doi: 10.1007/BF00252851.
6
Generation of coagulation factor V activity by cultured rabbit alveolar macrophages.培养的兔肺泡巨噬细胞产生凝血因子V活性。
J Exp Med. 1984 Dec 1;160(6):1880-90. doi: 10.1084/jem.160.6.1880.
7
Synthesis and turnover of prothrombin during experimental inflammation in rats.大鼠实验性炎症期间凝血酶原的合成与周转
Br J Exp Pathol. 1984 Dec;65(6):691-700.
8
Biosynthesis of coagulation Factor V by a human hepatocellular carcinoma cell line.人肝癌细胞系对凝血因子V的生物合成
J Clin Invest. 1984 Mar;73(3):654-8. doi: 10.1172/JCI111256.
9
Mechanism of action of vitamin K: evidence for the conversion of a precursor protein to prothrombin in the rat.维生素K的作用机制：大鼠体内前体蛋白转化为凝血酶原的证据。
Proc Natl Acad Sci U S A. 1971 Jul;68(7):1653-7. doi: 10.1073/pnas.68.7.1653.
10
Tissue factor activity in lymphocyte cultures from normal individuals and patients with hemophilia A.正常个体及甲型血友病患者淋巴细胞培养物中的组织因子活性。
J Clin Invest. 1973 Jun;52(6):1427-34. doi: 10.1172/JCI107316.

本文引用的文献

1
INHIBITION BY PUROMYCIN OF AMINO ACID INCORPORATION INTO PROTEIN.嘌呤霉素对氨基酸掺入蛋白质的抑制作用。
Proc Natl Acad Sci U S A. 1959 Dec;45(12):1721-9. doi: 10.1073/pnas.45.12.1721.
2
New one-stage procedures for the quantitative determination of prothrombin and labile factor.凝血酶原和不稳定因子定量测定的新型一步法
Am J Clin Pathol. 1950 Mar;20(3):233-40. doi: 10.1093/ajcp/20.3.233.
3
The dominant role of the liver in plasma protein synthesis; a direct study of the isolated perfused rat liver with the aid of lysine-epsilon-C14.肝脏在血浆蛋白合成中的主导作用；借助赖氨酸-ε-C14对离体灌注大鼠肝脏进行的直接研究。
J Exp Med. 1951 Nov;94(5):431-53. doi: 10.1084/jem.94.5.431.
4
The labile factor of prothrombin conversion: its consumption under normal and pathological conditions affecting blood coagulation.凝血酶原转化的不稳定因子：在影响血液凝固的正常和病理条件下其消耗情况。
J Clin Invest. 1951 Mar;30(3):252-62. doi: 10.1172/JCI102439.
5
Effect of removal of the liver on blood coagulation.肝脏切除对血液凝固的影响。
Am J Physiol. 1951 Jan;164(1):111-6. doi: 10.1152/ajplegacy.1950.164.1.111.
6
Plasma clotting factors in chronic hepatocellular disease.慢性肝细胞疾病中的血浆凝血因子。
N Engl J Med. 1960 Aug 11;263:278-82. doi: 10.1056/NEJM196008112630604.
7
Disappearance of factor V activity after incubation of plasm with heparin and heparinoids.血浆与肝素及类肝素孵育后因子V活性消失。
Thromb Diath Haemorrh. 1960 Jun 15;4:303-22.
8
PROTHROMBIN SYNTHESIS IN THE DOG.狗体内凝血酶原的合成
Am J Physiol. 1964 May;206:929-38. doi: 10.1152/ajplegacy.1964.206.5.929.
9
INHIBITION OF HEMOGLOBIN SYNTHESIS BY PUROMYCIN.嘌呤霉素对血红蛋白合成的抑制作用。
Biochemistry. 1963 Sep-Oct;2:1084-90. doi: 10.1021/bi00905a030.
10
NATURE OF PROTHROMBIN BIOSYNTHESIS: PREPROTHROMBINAEMIA IN VITAMIN K-DEFICIENCY.凝血酶原生物合成的本质：维生素K缺乏时的前凝血酶原血症
Nature. 1963 Nov 9;200:589-90. doi: 10.1038/200589a0.

Synthesis of clotting factors by the isolated perfused rat liver.

作者信息

Olson J P, Miller L L, Troup S B

出版信息

J Clin Invest. 1966 May;45(5):690-701. doi: 10.1172/JCI105384.

DOI:10.1172/JCI105384
PMID:5935358
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC292746/
Abstract
摘要