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人类角化棘皮瘤的自然消退

The spontaneous regression of keratoacanthoma in man.

作者信息

Ramselaar C G, van der Meer J B

出版信息

Acta Derm Venereol. 1976;56(3):245-51.

PMID:59511
Abstract

This study was undertaken to search for immune mediated mechanisms which could possibly be involved in the spontaneous regression of keratoacanthomas. In eleven patients with solitary keratoacanthoma, immunofluorescence studies did not reveal significant staining patterns, while skin tests, using autologous extracts of keratoacanthomas, were not compatible with true delayed cutaneous hypersensitivity reactions.

摘要

本研究旨在寻找可能参与角化棘皮瘤自发消退的免疫介导机制。在11例孤立性角化棘皮瘤患者中,免疫荧光研究未发现明显的染色模式,而使用角化棘皮瘤自体提取物进行的皮肤试验与真正的迟发性皮肤过敏反应不相符。

相似文献

1
The spontaneous regression of keratoacanthoma in man.人类角化棘皮瘤的自然消退
Acta Derm Venereol. 1976;56(3):245-51.
2
Non-immunological regression of dimethylbenz(A) anthracene-induced experimental keratoacanthomas in the rabbit.
Dermatologica. 1979;158(2):142-51. doi: 10.1159/000250755.
3
[The immune status of patients with solitary keratoacanthomas].[孤立性角化棘皮瘤患者的免疫状态]
Vestn Dermatol Venerol. 1988(6):37-41.
4
Immunological study of keratoacanthomas.
Arch Dermatol Res. 1980;268(2):149-55. doi: 10.1007/BF00403799.
5
Smooth muscle-associated antigen in experimental cutaneous squamous cell carcinoma, keratoacanthoma, and papilloma.实验性皮肤鳞状细胞癌、角化棘皮瘤和乳头状瘤中的平滑肌相关抗原。
Cancer Res. 1975 Dec;35(12):3741-5.
6
Immune response to human keratoacanthoma.对人类角化棘皮瘤的免疫反应。
Br J Dermatol. 1979 Dec;101(6):625-32. doi: 10.1111/j.1365-2133.1979.tb05640.x.
7
Keratoacanthoma: a clinically distinct variant of well differentiated squamous cell carcinoma.角化棘皮瘤:一种临床特征明显的高分化鳞状细胞癌变体。
Adv Anat Pathol. 1998 Sep;5(5):269-80.
8
Possible key role of granzyme B in keratoacanthoma regression.颗粒酶B在角化棘皮瘤消退中可能的关键作用。
Med Hypotheses. 2006;66(6):1129-32. doi: 10.1016/j.mehy.2005.12.035. Epub 2006 Feb 23.
9
Immunological reaction in keratoacanthoma, a spontaneously resolving skin tumor.角化棘皮瘤(一种可自发消退的皮肤肿瘤)中的免疫反应。
Cancer Res. 1973 Sep;33(9):2030-3.
10
[Cutaneous delayed hypersensitivity in a French reference population using the cell-mediated immunity multitest device].
Ann Anesthesiol Fr. 1981;22(3):270-8.

引用本文的文献

1
Keratoacanthoma.角化棘皮瘤
BMJ Case Rep. 2009;2009. doi: 10.1136/bcr.09.2008.0995. Epub 2009 Apr 28.
2
Loss of heterozygosity analysis of keratoacanthoma reveals multiple differences from cutaneous squamous cell carcinoma.角化棘皮瘤的杂合性缺失分析揭示了其与皮肤鳞状细胞癌的多处差异。
Br J Cancer. 1996 Mar;73(5):649-53. doi: 10.1038/bjc.1996.113.
3
Hypocomplementemia in keratoacanthoma.角化棘皮瘤中的补体低下
Arch Dermatol Res. 1981;270(2):189-91. doi: 10.1007/BF00408233.
4
Immunological study of keratoacanthomas.
Arch Dermatol Res. 1980;268(2):149-55. doi: 10.1007/BF00403799.
5
H-ras activation in benign and self-regressing skin tumors (keratoacanthomas) in both humans and an animal model system.人类和动物模型系统中良性及自行消退性皮肤肿瘤(角化棘皮瘤)中的H-ras激活。
Mol Cell Biol. 1988 Feb;8(2):786-93. doi: 10.1128/mcb.8.2.786-793.1988.
6
Oncogene activation in human benign tumors of the skin (keratoacanthomas): is HRAS involved in differentiation as well as proliferation?人类皮肤良性肿瘤(角化棘皮瘤)中的癌基因激活:HRAS是否参与分化以及增殖过程?
Proc Natl Acad Sci U S A. 1989 Aug;86(16):6372-6. doi: 10.1073/pnas.86.16.6372.
7
ras activation in human tumors and in animal model systems.人类肿瘤和动物模型系统中的Ras激活
Environ Health Perspect. 1991 Jun;93:19-25. doi: 10.1289/ehp.919319.