The chemistry of pain production.
作者信息
Keele C A
出版信息
Proc R Soc Med. 1967 Apr;60(4):419-22. doi: 10.1177/003591576706000434.
Abstract
摘要
相似文献
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The chemistry of pain production.疼痛产生的化学原理。
Proc R Soc Med. 1967 Apr;60(4):419-22. doi: 10.1177/003591576706000434.
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Influence of changed calcium and potassium concentration on the algesic effect of bradykinin and acetylcholine.钙和钾浓度变化对缓激肽和乙酰胆碱致痛作用的影响。
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5
Urticaria pigmentosa and factitious urticaria. Direct evidence for release of histamine and other smooth muscle-contracting agents in dermographic skin.色素性荨麻疹和人工性荨麻疹。皮肤划痕症皮肤中组胺及其他平滑肌收缩剂释放的直接证据。
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Evaluation of drugs applicable to treatment of migraine in the cranial circulation of the monkey.适用于治疗猴子颅内循环偏头痛的药物评估。
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Chemical response pattern of different classes of C-nociceptors to pruritogens and algogens.不同类别C型伤害感受器对致痒剂和致痛剂的化学反应模式。
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Stimulation of neovascularization of the cornea by biogenic amines.
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[Antagonistic relationship between anti-inflammatory and inflammatory agents in the skin].[皮肤中抗炎剂与炎症介质的拮抗关系]
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M-type K channels in peripheral nociceptive pathways.外周伤害感受通路上的 M 型 K 通道。
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Melittin, the Major Pain-Producing Substance of Bee Venom.蜂毒肽，蜂毒中主要的致痛物质。
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Potassium channels in peripheral pain pathways: expression, function and therapeutic potential.外周痛觉通路中的钾通道：表达、功能和治疗潜力。
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Activation of ATP-sensitive potassium channels antagonize nociceptive behavior and hyperexcitability of DRG neurons from rats.三磷酸腺苷敏感性钾通道的激活拮抗伤害感受行为和大鼠背根神经节神经元的过度兴奋。
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The nociceptive and anti-nociceptive effects of bee venom injection and therapy: a double-edged sword.蜂毒注射和疗法的痛觉和抗痛觉效应：一把双刃剑。
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The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl- channels.缓激肽在大鼠感觉神经元中诱导的急性伤害性信号是通过抑制 M 型钾通道和激活钙激活氯离子通道来介导的。
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Understanding inflammatory pain: ion channels contributing to acute and chronic nociception.理解炎症性疼痛：参与急性和慢性痛觉传递的离子通道。
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本文引用的文献
1
SEROTONIN--BRADYKININ POTENTIATION ON THE PAIN RECEPTORS IN MAN.血清素——缓激肽对人体痛觉感受器的增强作用。
Life Sci (1962). 1965 Feb;4:309-16. doi: 10.1016/0024-3205(65)90147-5.
2
PRODUCTION OF INFLAMMATORY CHANGES IN THE MICROCIRCULATION BY CATIONIC PROTEINS EXTRACTED FROM LYSOSOMES.从溶酶体中提取的阳离子蛋白引起微循环中的炎症变化。
J Exp Med. 1964 Nov 1;120(5):747-64. doi: 10.1084/jem.120.5.747.
3
A humoral agent implicated in vascular headache of the migraine type.一种与偏头痛型血管性头痛有关的体液因子。
Arch Neurol. 1960 Sep;3:223-9. doi: 10.1001/archneur.1960.00450030001001.
4
The toxin of Physalia nematocysts.
Ann N Y Acad Sci. 1960 Nov 17;90:742-50. doi: 10.1111/j.1749-6632.1960.tb26418.x.
5
Neurohumoral features of afferent fibers in man. Their role in vasodilation, inflammation, and pain.人类传入纤维的神经体液特征。它们在血管舒张、炎症和疼痛中的作用。
Arch Neurol. 1961 Jun;4:617-50. doi: 10.1001/archneur.1961.00450120031005.
6
A comparative study of kinin, kallidin, and bradykinin.激肽、胰激肽和缓激肽的比较研究。
Br J Pharmacol Chemother. 1957 Jun;12(2):149-58. doi: 10.1111/j.1476-5381.1957.tb00113.x.
7
The presence of histamine, 5-hydroxytryptamine and a potent, slow contracting substance in wasp venom.黄蜂毒液中存在组胺、5-羟色胺和一种强效的、作用缓慢的收缩物质。
Br J Pharmacol Chemother. 1954 Mar;9(1):53-8. doi: 10.1111/j.1476-5381.1954.tb00816.x.
8
Observations on chemical excitants of cutaneous pain in man.关于人类皮肤疼痛化学刺激物的观察
J Physiol. 1953 May 28;120(3):326-51. doi: 10.1113/jphysiol.1953.sp004898.
9
The Kallikrein-kininogen-kinin system.激肽释放酶-激肽原-激肽系统
Arthritis Rheum. 1966 Jun;9(3):473-82. doi: 10.1002/art.1780090311.
10
Urates and kinin formation in synovial fluid.滑液中尿酸盐和激肽的形成。
Proc R Soc Med. 1966 Apr;59(4):302-7. doi: 10.1177/003591576605900403.
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