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Acute pancreatic necrosis induced by puromycin.

作者信息

Longnecker D S, Farber E

出版信息

Lab Invest. 1967 Mar;16(3):321-9.

PMID:6023787
Abstract
摘要

相似文献

1
Acute pancreatic necrosis induced by puromycin.嘌呤霉素诱导的急性胰腺坏死
Lab Invest. 1967 Mar;16(3):321-9.
2
Molecular pathology of in-vivo inhibition of protein synthesis. Electron microscopy of rat pancreatic acinar cells in puromycin-induced necrosis.体内蛋白质合成抑制的分子病理学。嘌呤霉素诱导坏死的大鼠胰腺腺泡细胞的电子显微镜观察。
Am J Pathol. 1968 Apr;52(4):891-915.
3
Modification of puromycin-induced changes in pancreatic acinar cells by cycloheximide pretreatment in rats.
Lab Invest. 1972 Apr;26(4):459-64.
4
The effect of isoproterenol on amino acids in the submandibular gland.异丙肾上腺素对下颌下腺中氨基酸的影响。
Lab Invest. 1971 Sep;25(3):287-90.
5
Chemical pathology of acute amino acid deficiencies: morphologic and biochemical changes in young rats force-fed a threonine-deficienct diet.急性氨基酸缺乏的化学病理学：强制喂食苏氨酸缺乏饮食的幼鼠的形态学和生化变化。
J Nutr. 1968 Nov;96(3):349-58. doi: 10.1093/jn/96.3.349.
6
Incorporation of 1- 14 C-leucine into splenic and liver tissue of rats of different age.将1-¹⁴C-亮氨酸掺入不同年龄大鼠的脾脏和肝脏组织中。
Physiol Bohemoslov. 1971;20(4):291-5.
7
The use of chronically administered low doses of ethionine and puromycin to study increases in rat liver adenosine monophosphate deaminase activityused by thioacetamide injections.长期给予低剂量乙硫氨酸和嘌呤霉素以研究硫代乙酰胺注射所引起的大鼠肝脏腺苷单磷酸脱氨酶活性增加的用途。
Biochem Pharmacol. 1969 May;18(5):1083-91. doi: 10.1016/0006-2952(69)90112-9.
8
[In vitro incorporation of DL-1-leucine-C14 in to liver, kidney and diaphragm proteins of complete and orchiectomized male rats treated with DL-ethionine].[DL-乙硫氨酸处理的完整雄性大鼠和去势雄性大鼠肝脏、肾脏及膈肌蛋白质中DL-1-亮氨酸-C14的体外掺入]
Boll Soc Ital Biol Sper. 1968 Mar 31;44(6):525-8.
9
[Heterosexual and anabolic properties of methandrostenolone in experiments on female rats].
Probl Endokrinol Gormonoter. 1966 May-Jun;12(3):112-6.
10
Interrelationship between amino acid pools and protein synthesis in the rat submandibular gland.大鼠下颌下腺中氨基酸库与蛋白质合成之间的相互关系。
Biochim Biophys Acta. 1973 Jun 23;312(2):392-8. doi: 10.1016/0005-2787(73)90383-3.

引用本文的文献

1
Mechanisms of Pancreatic Injury Induced by Basic Amino Acids Differ Between L-Arginine, L-Ornithine, and L-Histidine.碱性氨基酸诱导胰腺损伤的机制在L-精氨酸、L-鸟氨酸和L-组氨酸之间存在差异。
Front Physiol. 2019 Jan 15;9:1922. doi: 10.3389/fphys.2018.01922. eCollection 2018.
2
Role of apoptosis in copper deficiency-induced pancreatic involution in the rat.细胞凋亡在大鼠铜缺乏诱导的胰腺退化中的作用。
Am J Pathol. 1993 Jun;142(6):1952-7.
3
Effect of cycloheximide on the cell cycle of the crypts of the small intestine of the rat.放线菌酮对大鼠小肠隐窝细胞周期的影响。
J Cell Biol. 1967 Dec;35(3):649-58. doi: 10.1083/jcb.35.3.649.
4
A light and electron microscope study of the exocrine pancreas following administration of 5-fluorouracil.5-氟尿嘧啶给药后外分泌胰腺的光镜和电镜研究
J Anat. 1969 Jan;104(Pt 1):93-108.
5
Molecular pathology of in-vivo inhibition of protein synthesis. Electron microscopy of rat pancreatic acinar cells in puromycin-induced necrosis.体内蛋白质合成抑制的分子病理学。嘌呤霉素诱导坏死的大鼠胰腺腺泡细胞的电子显微镜观察。
Am J Pathol. 1968 Apr;52(4):891-915.
6
Some observations on the acute histopathologic effects of cycloheximide in vivo.关于放线菌酮体内急性组织病理学效应的一些观察
Am J Pathol. 1971 Jan;62(1):111-25.
7
Hemolytic anemia and pancreatic acinar atrophy and fibrosis conditioned by "elemental" liquid diets and the ordinary intestinal microflora.由“要素”流质饮食和普通肠道微生物群引起的溶血性贫血以及胰腺腺泡萎缩和纤维化。
Ann Surg. 1971 Sep;174(3):469-510. doi: 10.1097/00000658-197109000-00016.
8
Mouse intestinal sensitivity to cycloheximide. A morphological, cytochemical and biochemical study.小鼠肠道对放线菌酮的敏感性：一项形态学、细胞化学和生物化学研究
Histochemie. 1971;27(1):50-62. doi: 10.1007/BF00263368.
9
Clinical effects of anticancer drugs to pancreatic diseases as protein synthesis inhibitors.
Gastroenterol Jpn. 1976;11(2):123-32. doi: 10.1007/BF02776709.
10
Prevention of the pancreatic complications after surgical procedures of the pancreas.
World J Surg. 1978 Nov;2(6):881-9. doi: 10.1007/BF01556548.