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Sodium transport and hypertension. Where are we going?

作者信息

Blaustein M P

出版信息

Hypertension. 1984 Jul-Aug;6(4):445-53. doi: 10.1161/01.hyp.6.4.445.

DOI:10.1161/01.hyp.6.4.445
PMID:6086515
Abstract
摘要

相似文献

1
Sodium transport and hypertension. Where are we going?钠转运与高血压。我们正走向何方？
Hypertension. 1984 Jul-Aug;6(4):445-53. doi: 10.1161/01.hyp.6.4.445.
2
[Intracellular electrolytes and high blood pressure. Membrane theory of essential hypertension].[细胞内电解质与高血压。原发性高血压的膜理论]
MMW Munch Med Wochenschr. 1983 Nov 11;125(45):1045-9.
3
Genetic and environmental factors influencing cellular sodium transport in essential hypertension.影响原发性高血压细胞钠转运的遗传和环境因素。
J Clin Hypertens. 1986 Jun;2(2):109-13.
4
Hypertension, non-insulin-dependent diabetes, and intracellular sodium metabolism.高血压、非胰岛素依赖型糖尿病与细胞内钠代谢
Hypertension. 1988 Mar;11(3):264-8. doi: 10.1161/01.hyp.11.3.264.
5
A role of calcium in altered sodium ion transport of hypertensives?
Life Sci. 1988;42(17):1573-81. doi: 10.1016/0024-3205(88)90435-3.
6
Familial aggregation of red blood cell cation transport systems in Japanese families.
Am J Epidemiol. 1985 Sep;122(3):386-90. doi: 10.1093/oxfordjournals.aje.a114119.
7
Membrane transport pathways and essential hypertension.
Scand J Clin Lab Invest Suppl. 1986;180:24-8.
8
[Active cationic flux and arterial hypertension].[活性阳离子通量与动脉高血压]
Med Clin (Barc). 1984 Oct 6;83(10):414-16.
9
Role of exogenous factors in alterations of red cell Na+-Li+ exchange and Na+-K+ cotransport in essential hypertension, primary hyperaldosteronism, and hypokalemia.外源性因素在原发性高血压、原发性醛固酮增多症和低钾血症患者红细胞钠-锂交换及钠-钾协同转运改变中的作用
Scand J Clin Lab Invest Suppl. 1986;180:82-95.
10
Cellular sodium transport in essential hypertension.
N Engl J Med. 1986 Jan 23;314(4):222-9. doi: 10.1056/NEJM198601233140407.

引用本文的文献

1
Na,K-ATPase as a target for endogenous cardiotonic steroids: What's the evidence?钠钾ATP酶作为内源性强心甾体的作用靶点：证据有哪些？
Genes Dis. 2020 Jan 22;8(3):259-271. doi: 10.1016/j.gendis.2020.01.008. eCollection 2021 May.
2
Increased sodium-dependent D-glucose transport in the jejunal brush-border membrane of spontaneously hypertensive rat.自发性高血压大鼠空肠刷状缘膜中钠依赖性D-葡萄糖转运增加。
Pflugers Arch. 1996 Jun;432(2):329-35. doi: 10.1007/s004240050140.
3
Alterations in sodium metabolism as an etiological model for hypertension.
作为高血压病因模型的钠代谢改变。
Cardiovasc Drugs Ther. 1995 Jun;9(3):377-99. doi: 10.1007/BF00879027.
4
Insulin does not regulate vascular smooth muscle Na+, K(+)-ATPase activity in rabbit aorta.胰岛素不调节兔主动脉血管平滑肌钠钾ATP酶活性。
Diabetologia. 1993 Mar;36(3):212-7. doi: 10.1007/BF00399952.
5
Black/white differences in response to antihypertensive therapy.抗高血压治疗反应中的黑/白差异。
J Natl Med Assoc. 1985 May;77 Suppl(Suppl):9-13.
6
Hypertension and sodium-lithium countertransport in Utah pedigrees: evidence for major-locus inheritance.犹他州家族中的高血压与钠-锂逆向转运：主基因遗传的证据
Am J Hum Genet. 1988 Jul;43(1):14-22.
7
[Hereditary salt sensitivity as a cause of essential hypertension: studies of membrane transport and intracellular electrolytes].
Klin Wochenschr. 1985 Sep 16;63(18):891-6. doi: 10.1007/BF01738142.
8
Sodium and hypertension. Still a controversy in 1986.钠与高血压。1986年这仍是一个有争议的问题。
Drugs. 1986;31 Suppl 4:29-39. doi: 10.2165/00003495-198600314-00005.
9
[Mechanism and significance of arteriolar media hypertrophy/ hyperplasia in arterial hypertension. Role of the Na+/H+ antiport].
Klin Wochenschr. 1988 Dec 1;66(23):1151-9. doi: 10.1007/BF01727661.
10
Spontaneously hypertensive rat vascular smooth muscle cells in culture exhibit increased growth and Na+/H+ exchange.培养的自发性高血压大鼠血管平滑肌细胞表现出生长增加和钠/氢交换增强。
J Clin Invest. 1989 Mar;83(3):822-9. doi: 10.1172/JCI113964.