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Influence of the converting enzyme inhibitors Hoe 498, enalapril and captopril on vascular reactivity of isolated arterial preparations.

作者信息

Schölkens B A, Xiang J Z, Tilly H

出版信息

Clin Exp Hypertens A. 1984;6(10-11):1807-13. doi: 10.3109/10641968409046083.

DOI:10.3109/10641968409046083
PMID:6099766
Abstract
摘要

相似文献

1
Influence of the converting enzyme inhibitors Hoe 498, enalapril and captopril on vascular reactivity of isolated arterial preparations.
Clin Exp Hypertens A. 1984;6(10-11):1807-13. doi: 10.3109/10641968409046083.
2
Inhibition of angiotensin converting enzyme by 2-[N-[(S)-1-carboxy-3-phenylpropyl]-L-alanyl]-(1S,3S,5S)-2-azabicyclo [3.3.0]octane-3-carboxylic acid (Hoe 498 diacid). Comparison with captopril and enalaprilat.2-[N-[(S)-1-羧基-3-苯基丙基]-L-丙氨酰]-(1S,3S,5S)-2-氮杂双环[3.3.0]辛烷-3-羧酸（Hoe 498二酸）对血管紧张素转换酶的抑制作用。与卡托普利和依那普利拉的比较。
Arzneimittelforschung. 1984;34(10B):1406-10.
3
Pharmacological properties of the new orally active angiotensin converting enzyme inhibitor 2-[N-[(S)-1-ethoxycarbonyl-3-phenylpropyl]-L-alanyl] -(1S,3S,5S)-2-azabicyclo[3.3.0]octane-3-carboxylic acid (Hoe 498).新型口服活性血管紧张素转换酶抑制剂2-[[(S)-1-乙氧羰基-3-苯基丙基]-L-丙氨酰基]-(1S,3S,5S)-2-氮杂双环[3.3.0]辛烷-3-羧酸（Hoe 498）的药理特性
Arzneimittelforschung. 1984;34(10B):1411-6.
4
[Comparisons of effects of Hoe-498 and enalapril in isolated guinea pig hearts and rat myocardium].[Hoe - 498与依那普利对豚鼠离体心脏和大鼠心肌作用的比较]
Zhongguo Yao Li Xue Bao. 1985 Jun;6(2):96-9.
5
2-[N-[(S)-1-ethoxycarbonyl-3-phenylpropyl]-L-alanyl]-(1S,3S,5S)-2- azabicyclo[3.3.0]octane-3-carboxylic acid (Hoe 498): antihypertensive action and persistent inhibition of tissue converting enzyme activity in spontaneously hypertensive rats.2-[N-[(S)-1-乙氧羰基-3-苯基丙基]-L-丙氨酰基]-(1S,3S,5S)-2-氮杂双环[3.3.0]辛烷-3-羧酸（Hoe 498）：对自发性高血压大鼠的降压作用及对组织转化酶活性的持续抑制
Arzneimittelforschung. 1984;34(10B):1426-30.
6
Unchanged pressor effect of norepinephrine in normal man following the oral administration of two angiotensin converting enzyme inhibitors, captopril and HOE 498.在正常人体内口服两种血管紧张素转换酶抑制剂卡托普利和HOE 498后去甲肾上腺素的升压作用未改变。
J Hypertens. 1986 Feb;4(1):9-11. doi: 10.1097/00004872-198602000-00002.
7
Is tissue converting enzyme inhibition a determinant of the antihypertensive efficacy of converting enzyme inhibitors? Studies with the two different compounds, Hoe498 and MK421, in spontaneously hypertensive rats.组织转换酶抑制作用是转换酶抑制剂降压疗效的决定因素吗？在自发性高血压大鼠中对两种不同化合物Hoe498和MK421的研究。
J Cardiovasc Pharmacol. 1984 Sep-Oct;6(5):872-80. doi: 10.1097/00005344-198409000-00021.
8
2-[N-[(S)-1-ethoxycarbonyl-3-phenylpropyl]-L-alanyl]-(1S,3S,5S) -2-azabicyclo[3.3.0]octane-3-carboxylic acid (Hoe 498)--a new and highly effective angiotensin I converting enzyme inhibitor.2-[N-[(S)-1-乙氧羰基-3-苯基丙基]-L-丙氨酰]-(1S,3S,5S)-2-氮杂双环[3.3.0]辛烷-3-羧酸（Hoe 498）——一种新型高效的血管紧张素I转换酶抑制剂。
Arzneimittelforschung. 1984;34(10B):1402-6.
9
Reduction of reperfusion arrhythmias in the ischemic isolated rat heart by angiotensin converting enzyme inhibitors: a comparison of captopril, enalapril, and HOE 498.
J Cardiovasc Pharmacol. 1986 Jul-Aug;8(4):722-8.
10
Comparison of the effects of the dipeptidyl peptidase inhibitors captopril, ramipril, and enalapril on water intake and sodium appetite of Sprague-Dawley rats.二肽基肽酶抑制剂卡托普利、雷米普利和依那普利对斯普拉格-道利大鼠饮水量和钠食欲影响的比较。
Behav Neurosci. 1988 Dec;102(6):953-60. doi: 10.1037//0735-7044.102.6.953.

引用本文的文献

1
Why are converting enzyme inhibitors vasodilators?为什么转换酶抑制剂是血管扩张剂？
Br J Clin Pharmacol. 1989;28 Suppl 2(Suppl 2):95S-103S; discussion 103S-104S. doi: 10.1111/j.1365-2125.1989.tb03585.x.
2
Comparisons in vitro, ex vivo, and in vivo of the actions of seven structurally diverse inhibitors of angiotensin converting enzyme (ACE).七种结构各异的血管紧张素转换酶（ACE）抑制剂作用的体外、离体和体内比较。
Br J Clin Pharmacol. 1989;28 Suppl 2(Suppl 2):115S-130S; discussion 130S-131S. doi: 10.1111/j.1365-2125.1989.tb03587.x.
3
Ramipril. A review of its pharmacological properties and therapeutic efficacy in cardiovascular disorders.
雷米普利。对其药理特性及在心血管疾病中的治疗效果的综述。
Drugs. 1990 Jan;39(1):110-35. doi: 10.2165/00003495-199039010-00009.