肌腱损伤时肌肉的缺血缺氧性改变（作者译）
[Ischemic-hypoxic changes in muscles with tendon injuries (author's transl)].
作者信息
Józsa L, Réffy A, Bálint B J, Järvinen M, Kvist M
出版信息
Magy Traumatol Orthop Helyreallito Seb. 1981;24(3):218-26.
PMID:6115992
Abstract
Ischemic-hypoxic changes in muscles with experimental tenotomy and human tendon ruptures have been examined. The so-called capillarization of the muscles is greatly reduced, following tendon injury and the accumulation of fibrous tissue is to be observed, around the muscle cells. The decreased blood supply and the elongation of the capillary muscle cell distance lead to hypoxic lesions (mitochondrial destruction, intramitochondrial calcification, accumulation of lysosomes etc.).
摘要
对实验性肌腱切断术和人类肌腱断裂后肌肉的缺血缺氧变化进行了研究。肌腱损伤后，所谓的肌肉毛细血管化显著减少，且在肌肉细胞周围可观察到纤维组织的堆积。血液供应减少以及毛细血管与肌肉细胞距离延长导致缺氧性病变（线粒体破坏、线粒体内钙化、溶酶体堆积等）。
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