Stovring J
J Comput Assist Tomogr. 1977 Jul;1(3):319-23. doi: 10.1097/00004728-197707000-00006.
Tentorial herniation secondary to supratentorial mass lesions will cause aqueductal compression and raised intraventricular pressure. Under ordinary circumstances, a raised intraventricular pressure will result in the development of hydrocephalus, but, in the presence of a large supratentorial mass, it is impossible for a generalized hydrocephalus to develop; only those parts of the ventricular system that are somehow shielded from the pressure effect of the mass lesion will be able to dilate. This dilatation will most frequently involve the contralateral temporal horn. Often, the atrium of the ventricle and the occipital horn will also be involved and, occasionally, the entire ventricle on the side opposite the mass will be dilated. The association of a large supratentorial mass with considerable shift of midline structures and computed tomography findings of a dilated contralateral temporal horn should be considered indicative of tentorial herniation, unless there are signs of preexisting atrophy or other preexisting disease processes that could have caused widening of the temporal horn.
幕上占位性病变继发的小脑幕切迹疝会导致导水管受压和脑室内压力升高。在通常情况下,脑室内压力升高会导致脑积水的发展,但是,在存在巨大幕上占位的情况下,不可能发生全身性脑积水;只有脑室系统中那些在某种程度上免受占位性病变压力影响的部分才能够扩张。这种扩张最常累及对侧颞角。通常,脑室的房部和枕角也会受累,偶尔,占位对侧的整个脑室会扩张。除非有先前存在萎缩或其他可能导致颞角增宽的疾病过程的迹象,否则巨大幕上占位与中线结构明显移位以及对侧颞角扩张的计算机断层扫描结果相关联应被视为小脑幕切迹疝的指征。