神经免疫性疾病的当前观点：多发性硬化症和实验性变态反应性脑脊髓炎(1,2)。 - Suppr

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超能文献

Current perspectives of neuroimmunologic disease: multiple sclerosis and experimental allergic encephalomyelitis (1,2).

作者信息

Paterson P Y, Day E D

出版信息

Clin Immunol Rev. 1981;1(4):581-697.

PMID:6188568

Abstract

摘要

相似文献

Current perspectives of neuroimmunologic disease: multiple sclerosis and experimental allergic encephalomyelitis (1,2).神经免疫性疾病的当前观点：多发性硬化症和实验性变态反应性脑脊髓炎(1,2)。

Clin Immunol Rev. 1981;1(4):581-697.

Conclusion: is myelin basic protein the right antigen and experimental allergic encephalomyelitis the right model for multiple sclerosis?结论：髓鞘碱性蛋白是正确的抗原，实验性自身免疫性脑脊髓炎是多发性硬化症的正确模型吗？

Prog Clin Biol Res. 1984;146:503-8.

A morphologic comparison of three experimental models of experimental allergic encephalomyelitis with multiple sclerosis.三种实验性变应性脑脊髓炎实验模型与多发性硬化症的形态学比较。

Prog Clin Biol Res. 1984;146:61-6.

Induction of experimental autoimmune encephalomyelitis in Lewis rats by a viral peptide with limited homology to myelin basic protein.用与髓鞘碱性蛋白具有有限同源性的病毒肽诱导Lewis大鼠实验性自身免疫性脑脊髓炎。

Exp Neurol. 2007 Aug;206(2):231-9. doi: 10.1016/j.expneurol.2007.04.015. Epub 2007 May 24.

Immunologic determinants of experimental neurologic autoimmune disease and approaches to the multiple sclerosis problem.实验性神经自身免疫性疾病的免疫决定因素及多发性硬化问题的解决方法。

Trans Am Clin Climatol Assoc. 1978;89:109-18.

Biology of disease. Analysis of autoimmune demyelination: its impact upon multiple sclerosis.疾病生物学。自身免疫性脱髓鞘分析：其对多发性硬化症的影响。

Lab Invest. 1984 Jun;50(6):608-35.

Experimental autoimmune demyelinating diseases: experimental allergic encephalomyelitis and experimental allergic neuritis.实验性自身免疫性脱髓鞘疾病：实验性变应性脑脊髓炎和实验性变应性神经炎。

Biomedicine. 1975 Mar;22(2):81-7.

Multiple sclerosis: immunopathologic mechanisms in the progression and resolution of inflammatory demyelination.多发性硬化症：炎症性脱髓鞘进展与消退中的免疫病理机制

Res Publ Assoc Res Nerv Ment Dis. 1990;68:37-54.

Immunopathogenic mechanisms in experimental allergic encephalomyelitis.实验性变态反应性脑脊髓炎中的免疫致病机制。

Curr Opin Neurol Neurosurg. 1993 Apr;6(2):182-8.

[Division of neuroimmunologic diseases. Foundation, principles, and classification].[神经免疫性疾病科。基础、原则与分类]

Nervenarzt. 1976 Jul;47(7):429-34.

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Direct binding of myelin basic protein and synthetic copolymer 1 to class II major histocompatibility complex molecules on living antigen-presenting cells--specificity and promiscuity.髓鞘碱性蛋白与合成共聚物1在活的抗原呈递细胞上与II类主要组织相容性复合体分子的直接结合——特异性与混杂性。

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Virology. 1984 Sep;137(2):347-57. doi: 10.1016/0042-6822(84)90227-7.

The clotting system: gatekeeper of cerebrovascular permeability and monitor of clinical manifestations of neuroautoimmune disease.凝血系统：脑血管通透性的守护者及神经自身免疫性疾病临床表现的监测者。

Trans Am Clin Climatol Assoc. 1986;97:149-57.

Induction of experimental allergic encephalomyelitis in Lewis rats with purified synthetic peptides: delineation of antigenic determinants for encephalitogenicity, in vitro activation of cellular transfer, and proliferation of lymphocytes.用纯化的合成肽诱导Lewis大鼠实验性变应性脑脊髓炎：致脑炎抗原决定簇的描绘、细胞转移的体外激活及淋巴细胞增殖

Proc Natl Acad Sci U S A. 1985 Aug;82(16):5515-9. doi: 10.1073/pnas.82.16.5515.

Simultaneous visualization of vascular permeability change and leukocyte egress in the central nervous system during autoimmune encephalomyelitis.自身免疫性脑脊髓炎期间中枢神经系统血管通透性变化和白细胞渗出的同步可视化。

Acta Neuropathol. 1987;74(2):191-3. doi: 10.1007/BF00692851.

Experimental allergic encephalomyelitis in rhesus monkeys: I. Immunological parameters in EAE resistant and susceptible rhesus monkeys.恒河猴实验性变应性脑脊髓炎：I. EAE抗性和易感性恒河猴的免疫学参数

Clin Exp Immunol. 1987 Apr;68(1):100-7.

Experimental allergic encephalomyelitis in the monkey: humoral immunity and blood-brain barrier function.猴实验性变应性脑脊髓炎：体液免疫与血脑屏障功能

Ital J Neurol Sci. 1989 Dec;10(6):561-5. doi: 10.1007/BF02333791.

Specific inhibition of the T-cell response to myelin basic protein by the synthetic copolymer Cop 1.合成共聚物Cop 1对T细胞针对髓鞘碱性蛋白反应的特异性抑制作用

Proc Natl Acad Sci U S A. 1988 Dec;85(24):9724-8. doi: 10.1073/pnas.85.24.9724.

Effect of rat and beta-human interferons on hyperacute experimental allergic encephalomyelitis in rats.

Agents Actions. 1985 Jul;16(5):397-403. doi: 10.1007/BF01982879.

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