Zavodskaia I S, Migas E A, Moreva E V
Farmakol Toksikol. 1984 Mar-Apr;47(2):23-8.
The development of experimental neurogenic gastric ulcers was followed by a reduction in the noradrenaline and cAMP levels in the gastric tissue. The energy deficiency and upset nucleic metabolism were shown to underlie the structural lesions of the gastric mucosa. L-DOPA and etimizol stimulating the metabolic processes and the function of the sympathetic nervous system intensify the reparative processes during gastric ulceration.
实验性神经源性胃溃疡形成后,胃组织中的去甲肾上腺素和环磷酸腺苷水平降低。能量缺乏和核酸代谢紊乱被证明是胃黏膜结构损伤的基础。左旋多巴和乙咪酯刺激代谢过程和交感神经系统功能,可增强胃溃疡形成过程中的修复过程。