Golenhofen K, Hannappel J
Experientia. 1978 Jan 15;34(1):64-5. doi: 10.1007/BF01921904.
In isolated muscle strips of porcine renal pelvis and ureter, the calcium antagonist nifedipine (3.10(-7) moles/l) completely suppressed spontaneous phasic mechanical activity and the phasic components of an adrenaline-induced activation (P-component). In the presence of nifedipine, adrenaline induced in pelvis preparations (but not in the ureter) a tonic contraction (T-component) which was on average 61% of the control reaction (SD +/- 26%; n = 35).
在猪肾盂和输尿管的离体肌肉条中，钙拮抗剂硝苯地平（3×10⁻⁷摩尔/升）完全抑制了自发性相性机械活动以及肾上腺素诱导激活的相性成分（P成分）。在硝苯地平存在的情况下，肾上腺素在肾盂制剂中（但不在输尿管中）诱导出强直性收缩（T成分），其平均为对照反应的61%（标准差±26%；n = 35）。
