Soria R, Fernandez F, Heller J, Brétille J, Cherif F, Barrillon A, Gerbaux A, Gay J
Arch Mal Coeur Vaiss. 1984 Dec;77(13):1468-80.
Forty-nine cases of Wolff-Parkinson-White syndrome (WPW) were diagnosed out of 10 750 patients with cardiac disease (0.45 p. 100), 24 cases out of 3 761 congenital malformations and 25 cases in the 6 989 patients with acquired heart disease. Right ventricular pre-excitation was recorded in 31 cases; 13 in the lateral zone, 12 in the posterior paraseptal zone and 6 in the anterior paraseptal zone. Left ventricular pre-excitation was recorded in 18 cases: 8 in the lateral zone, 5 in the anterior paraseptal and 5 in the posterior paraseptal zones. WPW and congenital heart disease: Out of 20 cases of Ebstein's anomaly, 5 cases of WPW were observed: 4 right posterior and 1 right lateral pre-excitations. Out of 218 cases of hypertrophic obstructive cardiomyopathy, 7 cases of WPW were observed, 4 of which were congenital. Three cases of WPW were recorded in 699 patients with ventricular septal defects. Out of 1 348 cases of atrial septal defect, 5 cases of pre-excitation were recorded, including 3 right posterior pre-excitations associated with an ostium primum defect. Pre-excitation was also observed in isolated cases of corrected transposition of the great arteries, supravalvular aortic stenosis, aortic incompetence and patent ductus arteriosus. Pre-excitation and acquired heart disease: Five cases of pre-excitation were recorded out of 305 cases of dilated cardiomyopathy (1.62 p. 100). Eleven cases of pre-excitation were recorded in a total of 3 471 cases of valvular heart disease (0.31 p. 100): 9 in rheumatic valve disease and 2 in mitral valve prolapse. Nine cases of pre-excitation were observed in 2 850 cases of coronary artery disease. Intermittent Wolff-Parkinson-White syndrome: Ventricular pre-excitation masks the ECG changes of complete right bundle branch block in Ebstein's anomaly, complete left bundle branch block in aortic incompetence and dilated cardiomyopathy, and the in-complete right bundle branch block often seen in mitral valve prolapse. The characteristic appearances of WPW depend on the zone of pre-excitation. Right ventricular hypertrophy observed in ventricular septal defect with pulmonary stenosis and mitral stenosis may be masked by right lateral pre-excitation. Changes of inferior wall myocardial infarction may be masked by left anterior wall pre-excitation. On the other hand, the effects of WPW on left ventricular hypertrophy are variable, high amplitudes of the resultant forces seeming to depend on late and isolated activation of one of the left ventricular walls.
在10750例心脏病患者中诊断出49例预激综合征（WPW）（每100例中有0.45例），在3761例先天性畸形患者中有24例，在6989例后天性心脏病患者中有25例。记录到右心室预激31例；其中13例位于外侧区，12例位于后间隔旁区，6例位于前间隔旁区。记录到左心室预激18例：8例位于外侧区，5例位于前间隔旁区，5例位于后间隔旁区。WPW与先天性心脏病：在20例埃布斯坦畸形患者中，观察到5例WPW：4例右后预激和1例右外侧预激。在218例肥厚性梗阻性心肌病患者中，观察到7例WPW，其中4例为先天性。在699例室间隔缺损患者中记录到3例WPW。在1348例房间隔缺损患者中，记录到5例预激，其中3例右后预激与原发孔缺损有关。在矫正型大动脉转位、主动脉瓣上狭窄、主动脉瓣关闭不全和动脉导管未闭的个别病例中也观察到预激。预激与后天性心脏病：在305例扩张型心肌病患者中记录到5例预激（每100例中有1.62例）。在总共3471例瓣膜性心脏病患者中记录到11例预激（每100例中有0.31例）：9例在风湿性瓣膜病中，2例在二尖瓣脱垂中。在2850例冠状动脉疾病患者中观察到9例预激。间歇性预激综合征：心室预激掩盖了埃布斯坦畸形中完全性右束支传导阻滞、主动脉瓣关闭不全和扩张型心肌病中完全性左束支传导阻滞以及二尖瓣脱垂中常见的不完全性右束支传导阻滞的心电图变化。WPW的特征性表现取决于预激区。室间隔缺损合并肺动脉狭窄和二尖瓣狭窄时观察到的右心室肥厚可能被右外侧预激掩盖。下壁心肌梗死的变化可能被左前壁预激掩盖。另一方面，WPW对左心室肥厚的影响是可变的，合力的高振幅似乎取决于左心室壁之一的晚期和孤立激活。
