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Kainic acid neurotoxicity toward hippocampal formation: dependence on specific excitatory pathways.

作者信息

Nadler J V, Cuthbertson G J

出版信息

Brain Res. 1980 Aug 11;195(1):47-56. doi: 10.1016/0006-8993(80)90865-3.

DOI:10.1016/0006-8993(80)90865-3
PMID:6249441
Abstract
摘要

相似文献

1
Kainic acid neurotoxicity toward hippocampal formation: dependence on specific excitatory pathways.海人酸对海马结构的神经毒性：对特定兴奋性通路的依赖性。
Brain Res. 1980 Aug 11;195(1):47-56. doi: 10.1016/0006-8993(80)90865-3.
2
Evidence from lesion studies for epileptogenic and non-epileptogenic neurotoxic interactions between kainic acid and excitatory innervation.来自损伤研究的证据表明，海藻酸与兴奋性神经支配之间存在致痫性和非致痫性神经毒性相互作用。
Brain Res. 1981 Feb 2;205(2):405-10. doi: 10.1016/0006-8993(81)90351-6.
3
Comparative toxicity of kainic acid and other acidic amino acids toward rat hippocampal neurons.
Neuroscience. 1981;6(12):2505-17. doi: 10.1016/0306-4522(81)90096-8.
4
Selective destruction by kainic acid of neurons innervated by putative glutamergic afferents in septum and nucleus of the diagonal band.
Brain Res. 1980 Jan 27;182(2):461-5. doi: 10.1016/0006-8993(80)91204-4.
5
Fate of the hippocampal mossy fiber projection after destruction of its postsynaptic targets with intraventricular kainic acid.用脑室注射红藻氨酸破坏海马苔藓纤维投射的突触后靶点后其命运
J Comp Neurol. 1981 Mar 10;196(4):549-69. doi: 10.1002/cne.901960404.
6
Role of excitatory pathways in the hippocampal damage produced by kainic acid.兴奋性通路在海藻酸所致海马损伤中的作用。
Adv Biochem Psychopharmacol. 1981;27:395-402.
7
Systemic injection of kainic acid: effect on neurotransmitter markers in piriform cortex, amygdaloid complex and hippocampus and protection by cortical lesioning and anticonvulsants.海人酸的全身注射：对梨状皮层、杏仁复合体和海马中神经递质标志物的影响以及皮层损伤和抗惊厥药的保护作用。
Neuroscience. 1982 May;7(5):1257-64. doi: 10.1016/0306-4522(82)91132-0.
8
Different sensitivity of hippocampal neurons to kainic acid in the rabbit.家兔海马神经元对 kainic 酸的不同敏感性。
Neuroscience. 1980;5(8):1474-8. doi: 10.1016/0306-4522(80)90009-3.
9
The role of epileptic activity in hippocampal and "remote" cerebral lesions induced by kainic acid.癫痫活动在由 kainic 酸诱导的海马和“远隔”脑损伤中的作用。
Brain Res. 1980 Jun 2;191(1):79-97. doi: 10.1016/0006-8993(80)90316-9.
10
Kainic acid induced seizures: neurochemical and histopathological changes.海藻酸诱发的癫痫发作：神经化学和组织病理学变化
Neuroscience. 1983 Dec;10(4):1301-15. doi: 10.1016/0306-4522(83)90113-6.

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The Future of Neurotoxicology: A Neuroelectrophysiological Viewpoint.神经毒理学的未来：神经电生理学视角
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Prosaposin, a neurotrophic factor, protects neurons against kainic acid-induced neurotoxicity.原朊素，一种神经营养因子，可保护神经元免受海人酸诱导的神经毒性。
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The matrix metalloproteinase inhibitor marimastat inhibits seizures in a model of kainic acid-induced status epilepticus.
基质金属蛋白酶抑制剂马立司他可抑制红藻氨酸诱导的癫痫持续状态模型中的癫痫发作。
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A Proline Derivative-Enriched Fraction from Protects the Hippocampus from Intracerebroventricular Pilocarpine-Induced Injury Associated with in Mice.一种富含脯氨酸衍生物的馏分通过抑制自噬对侧脑室注射匹罗卡品诱导的小鼠海马损伤发挥保护作用。
Int J Mol Sci. 2020 Jun 11;21(11):4188. doi: 10.3390/ijms21114188.
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A Model of Chronic Temporal Lobe Epilepsy Presenting Constantly Rhythmic and Robust Spontaneous Seizures, Co-morbidities and Hippocampal Neuropathology.一种呈现持续节律性且强烈自发癫痫发作、共病及海马神经病理学的慢性颞叶癫痫模型。
Aging Dis. 2019 Oct 1;10(5):915-936. doi: 10.14336/AD.2019.0720. eCollection 2019 Oct.
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Interneurons secrete prosaposin, a neurotrophic factor, to attenuate kainic acid-induced neurotoxicity.中间神经元分泌神经营养因子prosaposin，以减轻海藻酸诱导的神经毒性。
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Effects on Spatial Cognition and Nociceptive Behavior Following Peripheral Nerve Injury in Rats with Lesion of the Striatal Marginal Division Induced by Kainic Acid.海人酸诱导纹状体边缘区损伤的大鼠外周神经损伤后对空间认知和伤害性感受行为的影响
Neurochem Res. 2015 Nov;40(11):2357-64. doi: 10.1007/s11064-015-1727-6. Epub 2015 Sep 28.
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A prosaposin-derived Peptide alleviates kainic Acid-induced brain injury.一种源自prosaposin的肽可减轻海藻酸诱导的脑损伤。
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Prosaposin overexpression following kainic acid-induced neurotoxicity.海藻酸诱导的神经毒性后prosaposin的过表达。
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Susceptibility to seizure-induced excitotoxic cell death is regulated by an epistatic interaction between Chr 18 (Sicd1) and Chr 15 (Sicd2) loci in mice.小鼠癫痫诱导的兴奋性毒性细胞死亡易感性受18号染色体（Sicd1）和15号染色体（Sicd2）位点之间上位相互作用的调节。
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