前列环素对血小板活化因子诱导的兔血小板聚集的影响。
Effect of prostacyclin on platelet-activating factor induced rabbit and platelet aggregation.
作者信息
Bussolino F, Camussi G
出版信息
Prostaglandins. 1980 Nov;20(5):781-91. doi: 10.1016/0090-6980(80)90133-1.
PMID:6258196
Abstract
In this paper, the effect of prostacyclin (PGI2) on the aggregation induced by Platelet-activating factor (PAF), a phospholipid mediator of anaphylaxis, was studied. Synthetic PGI2 and PGI2-like activity generated from rabbit aorta were demonstrated to be effective inhibitors of PAF-induced rabbit platelet aggregation and release of 3H-serotonin (3H-5HT).
摘要
本文研究了前列环素（PGI2）对血小板活化因子（PAF）诱导的聚集的影响，PAF是一种过敏反应的磷脂介质。合成的PGI2和从兔主动脉产生的PGI2样活性被证明是PAF诱导的兔血小板聚集和3H-血清素（3H-5HT）释放的有效抑制剂。
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