Gilden D H, Devlin M, Wroblewska Z
Arch Virol. 1981;67(2):181-5. doi: 10.1007/BF01318603.
Superinfection of visna virus (VV)-infected cells with vesicular stomatitis virus (VSV) resulted in the formation of a pseudotype virus population containing a VSV genome within a VV coat [designated VSV (VV)] as determined by plaque reduction neutralization with antisera to VSV and VV. These VSV (VV) virions were capable of infecting cell cultures from a number of species that were non-permissive for VV alone. Limited propagation of VV in some mammalian species would thus appear to be due to an intracellular restriction rather than to absence of VV receptors.
用水疱性口炎病毒（VSV）对维斯纳病毒（VV）感染的细胞进行双重感染，结果形成了一种假型病毒群体，其在VV衣壳内含有VSV基因组[命名为VSV（VV）]，这是通过用抗VSV和VV的抗血清进行蚀斑减少中和试验确定的。这些VSV（VV）病毒粒子能够感染来自许多物种的细胞培养物，而这些细胞培养物单独对VV是不敏感的。因此，VV在某些哺乳动物物种中的有限增殖似乎是由于细胞内限制，而不是由于缺乏VV受体。
