人早幼粒细胞白血病细胞系（HL60）中趋化性和甲酰肽结合的发展。 - Suppr

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超能文献

Development of chemotaxis and formyl peptide binding in human promyelocytic leukemia cell line (HL60).

作者信息

Wood P A, Douglas S D

出版信息

Proc Soc Exp Biol Med. 1982 Apr;169(4):421-6. doi: 10.3181/00379727-169-41369.

DOI:10.3181/00379727-169-41369

PMID:6280196

Abstract

摘要

相似文献

Development of chemotaxis and formyl peptide binding in human promyelocytic leukemia cell line (HL60).人早幼粒细胞白血病细胞系（HL60）中趋化性和甲酰肽结合的发展。

Proc Soc Exp Biol Med. 1982 Apr;169(4):421-6. doi: 10.3181/00379727-169-41369.

The neutrophil N-formyl peptide receptor: dynamics of ligand-receptor interactions and their relationship to cellular responses.中性粒细胞N-甲酰基肽受体：配体-受体相互作用的动力学及其与细胞反应的关系。

Contemp Top Immunobiol. 1984;14:29-82. doi: 10.1007/978-1-4757-4862-8_2.

1,25-Dihydroxyvitamin D3-induced differentiation in a human promyelocytic leukemia cell line (HL-60): receptor-mediated maturation to macrophage-like cells.1,25-二羟基维生素D3诱导人早幼粒细胞白血病细胞系（HL-60）分化：受体介导的向巨噬细胞样细胞的成熟。

J Cell Biol. 1984 Feb;98(2):391-8. doi: 10.1083/jcb.98.2.391.

Human neutrophil Fc gamma RIIIB and formyl peptide receptors are functionally linked during formyl-methionyl-leucyl-phenylalanine-induced chemotaxis.在甲酰甲硫氨酰亮氨酰苯丙氨酸诱导的趋化作用过程中，人中性粒细胞FcγRIIIB和甲酰肽受体在功能上相互关联。

J Immunol. 1992 Aug 1;149(3):989-97.

Neutrophil chemoattractant fMet-Leu-Phe receptor expression and ionic events following activation.活化后中性粒细胞趋化因子N-甲酰甲硫氨酰-亮氨酰-苯丙氨酸受体的表达及离子事件

Contemp Top Immunobiol. 1984;14:83-108. doi: 10.1007/978-1-4757-4862-8_3.

Sulfasalazine inhibition of binding of N-formyl-methionyl-leucyl-phenylalanine (FMLP) to its receptor on human neutrophils.柳氮磺胺吡啶对N-甲酰甲硫氨酰亮氨酰苯丙氨酸（FMLP）与其在人中性粒细胞上的受体结合的抑制作用。

Biochem Pharmacol. 1984 Feb 1;33(3):407-12. doi: 10.1016/0006-2952(84)90233-8.

Modulation of PMN receptors for chemotactic peptides.趋化肽的中性粒细胞受体调节

Surv Immunol Res. 1983;2(2):139-44. doi: 10.1007/BF02918572.

Reduced chemotactic peptide binding in juvenile periodontitis: a model for neutrophil function.青少年牙周炎中趋化肽结合减少：中性粒细胞功能的一个模型

Biochem Biophys Res Commun. 1981 Jun 16;100(3):1278-84. doi: 10.1016/0006-291x(81)91962-8.

Removal of human polymorphonuclear leukocyte surface sialic acid inhibits reexpression (or recycling) of formyl peptide receptors. A possible explanation for its effect on formyl peptide-induced polymorphonuclear leukocyte chemotaxis.去除人多形核白细胞表面唾液酸会抑制甲酰肽受体的重新表达（或再循环）。这可能是其对甲酰肽诱导的多形核白细胞趋化作用的一种解释。

J Immunol. 1987 Sep 15;139(6):1978-84.

Effects of tunicamycin on the expression and function of formyl peptide chemotactic receptors of differentiated HL-60 cells.衣霉素对分化的HL-60细胞甲酰肽趋化受体表达及功能的影响。

J Immunol. 1986 Jun 15;136(12):4623-30.

引用本文的文献

J Cell Biol. 1984 Feb;98(2):391-8. doi: 10.1083/jcb.98.2.391.

Cytostasis of tumor cell lines by promyelocytic leukemia cell line HL60 differentiated to granulocyte lineage.向粒细胞系分化的早幼粒细胞白血病细胞系HL60对肿瘤细胞系的细胞生长抑制作用。

J Cancer Res Clin Oncol. 1985;109(2):103-6. doi: 10.1007/BF00391883.

Effect of pertussis toxin and neomycin on G-protein-regulated polyphosphoinositide phosphodiesterase. A comparison between HL60 membranes and permeabilized HL60 cells.百日咳毒素和新霉素对G蛋白调节的多磷酸肌醇磷酸二酯酶的作用。HL60细胞膜与透化HL60细胞的比较。

Biochem J. 1988 Dec 1;256(2):343-50. doi: 10.1042/bj2560343.

Guanine nucleotides stimulate polyphosphoinositide phosphodiesterase and exocytotic secretion from HL60 cells permeabilized with streptolysin O.鸟嘌呤核苷酸刺激用链球菌溶血素O通透处理的HL60细胞中的多磷酸肌醇磷酸二酯酶和胞吐分泌。

Biochem J. 1988 Mar 1;250(2):375-82. doi: 10.1042/bj2500375.

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