Bonazzi L, Raise E, Cuccí A M, Ruffilli E, Lenzi G, Di Salvo S, Pittralis M C
Minerva Med. 1982 Apr 28;73(18):1161-8.
The Authors analyse the results of their tryls about the markers of HBV and HAV acute hepatitis (HBsAg, anti-HAVAb) by R.I.Z. method. HAVAb was in 75% of the cases, its meaning was of post-contact with HAV. The title of HAVAb was effected in 18 patients with viral acute hepatitis; the results were 3 cases of HAV acute hepatitis and in other 7 cases no Ano B viral acute hepatitis. The 58.3% of acute viral hepatitis was HBsAg positive, the study of other markers of HBV and the title of HAVAb showed a viral acute hepatitis caused by HBV. We were not able to demonstrate the viruses which caused 7 HBsAg negative acute virale hepatitis, anti-HBcAg was in 97.8% of HBV acute hepatitis; its the most sensitive of HAV past-infection. The system c-anti-e was in 78.2% of HBsAg viral acute hepatitis. The persistence after 7th week of illness of HBeAg coincided with the hepatitis cronicity. On the contrary anti-HBeAg has not always a protective meaning.
作者通过R.I.Z.方法分析了他们关于乙肝病毒(HBV)和甲型肝炎病毒(HAV)急性肝炎标志物(HBsAg、抗-HAVAb)的试验结果。抗-HAVAb在75%的病例中出现,其意义为曾接触过HAV。抗-HAVAb滴度在18例病毒性急性肝炎患者中受到影响;结果为3例HAV急性肝炎,另外7例无乙型病毒性急性肝炎。58.3%的急性病毒性肝炎HBsAg呈阳性,对HBV其他标志物及抗-HAVAb滴度的研究显示为HBV引起的病毒性急性肝炎。我们未能证实导致7例HBsAg阴性急性病毒性肝炎的病毒,抗-HBcAg在97.8%的HBV急性肝炎中出现;它是既往感染HAV最敏感的指标。抗-HBe系统在78.2%的HBsAg病毒性急性肝炎中出现。病程第7周后HBeAg持续存在与肝炎慢性化相符。相反,抗-HBeAg并非总是具有保护意义。