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Activation of cellular stress protein genes by herpes simplex virus temperature-sensitive mutants which overproduce immediate early polypeptides.

作者信息

Notarianni E L, Preston C M

出版信息

Virology. 1982 Nov;123(1):113-22. doi: 10.1016/0042-6822(82)90299-9.

DOI:10.1016/0042-6822(82)90299-9
PMID:6293187
Abstract
摘要

相似文献

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Activation of cellular stress protein genes by herpes simplex virus temperature-sensitive mutants which overproduce immediate early polypeptides.单纯疱疹病毒温度敏感突变体对细胞应激蛋白基因的激活作用，这些突变体过量产生即刻早期多肽。
Virology. 1982 Nov;123(1):113-22. doi: 10.1016/0042-6822(82)90299-9.
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Identification of a herpes simplex virus function that represses late gene expression from parental viral genomes.鉴定一种抑制亲本病毒基因组晚期基因表达的单纯疱疹病毒功能。
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Co-ordinate regulation of herpes simplex virus gene expression is mediated by the functional interaction of two immediate early gene products.单纯疱疹病毒基因表达的协同调控是由两种立即早期基因产物的功能相互作用介导的。
J Mol Biol. 1986 Oct 5;191(3):395-409. doi: 10.1016/0022-2836(86)90135-x.

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Repression of gene expression upon infection of cells with herpes simplex virus type 1 mutants impaired for immediate-early protein synthesis.感染单纯疱疹病毒1型突变体（这些突变体在立即早期蛋白合成方面存在缺陷）后，细胞中基因表达受到抑制。
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Cellular stress inhibits transposition of the yeast retrovirus-like element Ty3 by a ubiquitin-dependent block of virus-like particle formation.细胞应激通过泛素依赖性阻断病毒样颗粒形成来抑制酵母逆转录病毒样元件Ty3的转座。
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