Schmid C, Steiner T, Froesch E R
FEBS Lett. 1982 Nov 1;148(1):31-4. doi: 10.1016/0014-5793(82)81236-2.
Parathyroid hormone stimulates [U-14C]glucose incorporation into glycogen of cultured osteoblast-like calvaria cells. This effect is detectable only several hours after the addition of PTH and it is mimicked by dibutyryl cyclic AMP. In contrast to insulin (in pharmacological concentrations), PTH enhances glycogen formation only in calvaria cells, but not in fibroblasts. Insulin-like growth factor I in physiological concentrations promotes glycogen-synthesis shortly after addition.
甲状旁腺激素刺激[U-14C]葡萄糖掺入培养的成骨样颅骨细胞的糖原中。这种作用仅在添加甲状旁腺激素数小时后才可检测到,并且二丁酰环磷酸腺苷可模拟这种作用。与胰岛素(药理浓度)不同,甲状旁腺激素仅增强颅骨细胞中的糖原形成,而不增强成纤维细胞中的糖原形成。生理浓度的胰岛素样生长因子I在添加后不久可促进糖原合成。