猴疱疹病毒非致癌变体中DNA序列的缺失。
Deletion of DNA sequence in a nononcogenic variant of Herpesvirus saimiri.
作者信息
Koomey J M, Mulder C, Burghoff R L, Fleckenstein B, Desrosiers R C
出版信息
J Virol. 1984 May;50(2):662-5. doi: 10.1128/JVI.50.2.662-665.1984.
Abstract
The 110-kilobase-pair stretch of unique sequence DNA of Herpesvirus saimiri is flanked by highly repetitive DNA. Detailed restriction endonuclease mapping has localized the left junction of repetitive and unique DNA to a 100-base-pair region. H. saimiri 11att, a replication competent nononcogenic variant of strain 11, has a deletion of 2.3 kilobase pairs of sequence information that spans this left junction of repetitive and unique DNA.
摘要
赛米里疱疹病毒110千碱基对的独特序列DNA片段两侧是高度重复的DNA。详细的限制性内切酶图谱已将重复DNA和独特DNA的左连接点定位到一个100碱基对的区域。赛米里疱疹病毒11att是11型的一种具有复制能力的非致癌变体，它缺失了2.3千碱基对的序列信息，该序列信息跨越了重复DNA和独特DNA的这个左连接点。
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