利用H-2突变体分析细胞毒性T淋巴细胞反应的特异性与调控。 - Suppr

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Specificity and regulation of cytotoxic T-lymphocyte responses analyzed with H-2 mutants.

作者信息

Melief C J, Stukart M J, de Waal L P, Kast W M, Melvold R W

出版信息

Transplant Proc. 1983 Dec;15(4):2086-9.

PMID:6324427

Abstract

摘要

相似文献

Specificity and regulation of cytotoxic T-lymphocyte responses analyzed with H-2 mutants.利用H-2突变体分析细胞毒性T淋巴细胞反应的特异性与调控。

Transplant Proc. 1983 Dec;15(4):2086-9.

Regulation of the cytotoxic T lymphocyte response against Sendai virus analyzed with H-2 mutants.用H-2突变体分析针对仙台病毒的细胞毒性T淋巴细胞反应的调控。

J Immunol. 1983 Mar;130(3):1090-6.

T lymphocyte responses to multiple minor histocompatibility antigens generate both self-major histocompatibility complex-restricted and cross-reactive cytotoxic T lymphocytes.T淋巴细胞对多种次要组织相容性抗原的反应会产生自身主要组织相容性复合体限制的和交叉反应性细胞毒性T淋巴细胞。

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A crucial role of the H-2 D locus in the regulation of both the D- and the K-associated cytotoxic T lymphocyte response against Moloney leukemia virus, demonstrated with two Db mutants.利用两个Db突变体证明了H-2 D基因座在调节针对莫洛尼白血病病毒的D相关和K相关细胞毒性T淋巴细胞反应中的关键作用。

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Clonal analysis of the T lymphocyte response to murine sarcoma virus-induced tumors.

Transplant Proc. 1982 Sep;14(3):556-8.

Linked help in the cytotoxic T-cell response revealed by adoptive transfer.通过过继转移揭示的细胞毒性T细胞反应中的连锁帮助。

Transplant Proc. 1983 Dec;15(4):2121-4.

Flow-microfluorometric monitoring of oligoclonal CD8+ T cell responses to an immunodominant Moloney leukemia virus-encoded epitope in vivo.体内寡克隆CD8 + T细胞对莫洛尼白血病病毒编码的免疫显性表位反应的流式微量荧光监测。

J Immunol. 1998 Feb 15;160(4):1659-65.

Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. I. H-2-encoded dominant gene control.对AKR/Gross病毒性白血病细胞溶解性T淋巴细胞反应诱导的遗传控制。I. H-2编码的显性基因控制。

J Immunol. 1984 May;132(5):2658-64.

Genetic control of the induction of cytolytic T lymphocyte responses to AKR/Gross viral leukemias. II. Negative control by the Fv-1 locus in AKR mice of responder H-2b haplotype.对AKR/Gross病毒性白血病细胞毒性T淋巴细胞反应诱导的遗传控制。II. 应答性H-2b单倍型AKR小鼠中Fv-1基因座的负调控。

J Immunol. 1984 May;132(5):2665-71.

Viral cross-reactivity and antigenic determinants recognized by human parainfluenza virus type 1-specific cytotoxic T-cells.1型人副流感病毒特异性细胞毒性T细胞识别的病毒交叉反应性和抗原决定簇

Virology. 1994 Mar;199(2):376-83. doi: 10.1006/viro.1994.1135.

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Three spontaneous H-2Db mutants are generated by genetic micro-recombination (gene conversion) events. Impact on the H-2-restricted immune responsiveness.通过基因微重组（基因转换）事件产生了三个自发的H-2Db突变体。对H-2限制性免疫反应性的影响。

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Cooperation between cytotoxic and helper T lymphocytes in protection against lethal Sendai virus infection. Protection by T cells is MHC-restricted and MHC-regulated; a model for MHC-disease associations.细胞毒性T淋巴细胞与辅助性T淋巴细胞在抵抗致死性仙台病毒感染中的协同作用。T细胞介导的保护作用受主要组织相容性复合体（MHC）限制并受其调控；一种MHC与疾病关联的模型。

J Exp Med. 1986 Sep 1;164(3):723-38. doi: 10.1084/jem.164.3.723.

Friend virus-specific cytotoxic T lymphocytes recognize both gag and env gene-encoded specificities.Friend病毒特异性细胞毒性T淋巴细胞识别gag和env基因编码的特异性。

J Exp Med. 1986 Jul 1;164(1):211-26. doi: 10.1084/jem.164.1.211.

Biochemical analysis of related, independently arising histocompatibility mutants: bm17 and KB-98 enlarge the "bg series" of H-2Kb mutants.相关的、独立产生的组织相容性突变体的生化分析：bm17和KB-98扩展了H-2Kb突变体的“bg系列”。

Biochem Genet. 1990 Aug;28(7-8):433-41. doi: 10.1007/BF02401430.

A cluster of mutations in HLA-A2 alpha 2 helix abolishes peptide recognition by T cells.

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