鼠伤寒沙门氏菌的厌氧培养物不表现出recA基因的可诱导蛋白水解功能和recBC功能。
Anaerobic cultures of Salmonella typhimurium do not exhibit inducible proteolytic function of the recA gene and recBC function.
作者信息
Droffner M L, Yamamoto N
出版信息
J Bacteriol. 1983 Nov;156(2):962-5. doi: 10.1128/jb.156.2.962-965.1983.
Abstract
In a strict anaerobic environment, lack of expression of bacterial recBC function and recA regulatory functions for the SOS repair system is demonstrated by the use of the carcinogenesis-mutagenesis assay and Salmonella phage P22 mutants requiring these host functions for replication. Therefore, we suggest that error-prone repair (SOS repair) is confined to aerobic environments in facultative anaerobes.
摘要
在严格的厌氧环境中，通过致癌-诱变试验以及对复制需要这些宿主功能的沙门氏菌噬菌体P22突变体的使用，证明了细菌recBC功能和SOS修复系统的recA调节功能缺乏表达。因此，我们认为易错修复（SOS修复）局限于兼性厌氧菌的需氧环境中。
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