缺乏前列腺素内源性前体的大鼠炎症模型。
Inflammatory models in rats depleted of endogenous precursors of prostaglandins.
作者信息
Bonta I L, Bult H, Parnham M J, Vincent J E
出版信息
Agents Actions. 1978 Jan;8(1-2):98-101. doi: 10.1007/BF01972409.
PMID:636960
Abstract
Inflammatory models in rats, depleted of prostaglandin precursors, are discussed as a novel approach to investigate anti-inflammatory drug mechanisms independent of interaction with the production of prostaglandins and to study the ambivalent (pro- or anti-inflammatory) role of endogenous prostaglandins.
摘要
在大鼠中，前列腺素前体被耗尽的炎症模型被作为一种新方法来讨论，该方法用于研究独立于与前列腺素产生相互作用的抗炎药物机制，并研究内源性前列腺素的矛盾（促炎或抗炎）作用。
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