Sibony P A, Lessell S, Gittinger J W
Surv Ophthalmol. 1984 Mar-Apr;28(5):382-90. doi: 10.1016/0039-6257(84)90243-1.
Paradoxical patterns of pupillary, lid and eye movement may follow oculomotor nerve palsy or they can develop spontaneously in patients with no known history of oculomotor palsy. The mechanism of this condition, known variously as aberrant regeneration of the third nerve, oculomotor misdirection or acquired oculomotor synkinesis, is not known, although the prevailing opinion has held that it occurs when axons regenerating within an oculomotor nerve become misdirected and innervate muscles for which they were not intended. However, there is evidence against this hypothesis. The authors critically review the various hypotheses and elucidate the controversy concerning the pathogenesis of acquired oculomotor synkinesis.
瞳孔、眼睑及眼球运动的异常模式可能继发于动眼神经麻痹，也可能在无动眼神经麻痹病史的患者中自发出现。这种情况有多种称谓，如动眼神经异常再生、动眼神经错向或后天性动眼神经联合运动，其机制尚不清楚，尽管普遍观点认为，当动眼神经内再生的轴突发生错向并支配非其原计划支配的肌肉时，就会出现这种情况。然而，有证据反对这一假说。作者对各种假说进行了批判性回顾，并阐明了关于后天性动眼神经联合运动发病机制的争议。
