产时母体过度通气作为胎儿心动过速的可能原因。一项临床与实验研究。

Maternal hyperventilation as a possible cause of fetal tachycardia sub partu. A clinical and experimental study.

作者信息

Müller G, Huber J C, Salzer H, Reinold E

出版信息

Gynecol Obstet Invest. 1984;17(5):270-5. doi: 10.1159/000299161.

Abstract

Examination of 28 agitated, hyperventilating patients with fetal tachycardia showed that the mothers were in a hyperventilation-related state of hypocapnia and alkalosis. The mean maternal pCO2 was 17.48 +/- 6.79 mm Hg, the pH 7.54 +/- 0.14, which caused a reflex spasm of the umbilical veins, fetal acidosis and fetal tachycardia. Additional experiments on sheep revealed a similar correlation between maternal hyperventilation, hypocapnia, respiratory alkalosis and fetal tachycardia while the flow volume of the uterus showed no change.

摘要

对28名烦躁不安、呼吸急促且伴有胎儿心动过速的患者进行检查发现，这些母亲处于与过度通气相关的低碳酸血症和碱中毒状态。母亲的平均动脉血二氧化碳分压为17.48±6.79毫米汞柱，pH值为7.54±0.14，这导致脐静脉反射性痉挛、胎儿酸中毒和胎儿心动过速。对绵羊进行的额外实验显示，母亲过度通气、低碳酸血症、呼吸性碱中毒与胎儿心动过速之间存在类似的相关性，而子宫血流量没有变化。