迷走神经对胆囊收缩的控制。
Vagal control of gall-bladder contraction.
作者信息
Magee D F, Naruse S, Pap A
出版信息
J Physiol. 1984 Oct;355:65-70. doi: 10.1113/jphysiol.1984.sp015406.
Abstract
The role of the vagi in gall-bladder contraction was studied in conscious dogs with chronic biliary fistulae. The interdigestive periodic contraction of this organ was abolished by bilateral cervical vagal blockade, atropine or pentolinium, but neither methacholine nor carbachol initiated contraction when the duodenum was kept alkaline. The orthodox view that gall-bladder evacuation is primarily controlled by humoral mechanisms needs re-evaluation. Vagal blockade prevented the usual gall-bladder contraction in response to duodenal acidification.
摘要
在患有慢性胆瘘的清醒犬中研究了迷走神经在胆囊收缩中的作用。该器官的消化间期周期性收缩可通过双侧颈迷走神经阻滞、阿托品或潘托铵消除，但当十二指肠保持碱性时，乙酰甲胆碱和卡巴胆碱均不能引发收缩。胆囊排空主要由体液机制控制这一传统观点需要重新评估。迷走神经阻滞可防止胆囊对十二指肠酸化产生通常的收缩反应。
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