Piatti L, Locatelli V, Ferracini C, Sozzi G
G Ital Cardiol. 1984 Aug;14(8):602-5.
A 34-year-old woman without heart disease had an acute pulmonary edema after a grand mal seizure. Neurogenic pulmonary edema is characterized by the primary role played by the central nervous system in its pathogenesis, usually in the absence of cardiopulmonary disease. We believe that the initiating mechanism was a marked, although brief, generalized vasoconstriction, followed by a shift of blood from the peripheral vascular bed to the pulmonary vascular bed. We stress the need of treating the underlying neurologic disease as the only regional way of preventing the recurrence of pulmonary edema.
一名无心脏病史的34岁女性在癫痫大发作后出现急性肺水肿。神经源性肺水肿的特征是中枢神经系统在其发病机制中起主要作用，通常发生在无心肺疾病的情况下。我们认为起始机制是显著的，尽管短暂的全身性血管收缩，随后血液从外周血管床转移至肺血管床。我们强调将基础神经疾病作为预防肺水肿复发的唯一有效方法进行治疗的必要性。
