Animal model of human disease. Increased sensitivity to ozone. Animal model: mice with low levels of G-6-PD.
作者信息
Calabrese E J
出版信息
Am J Pathol. 1978 May;91(2):409-12.
Abstract
摘要
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The effect of in vivo ozone exposure to Dorset sheep, an animal model with low levels of erythrocyte glucose-6-phosphate dehydrogenase activity.体内臭氧暴露对多塞特羊（一种红细胞葡萄糖-6-磷酸脱氢酶活性水平较低的动物模型）的影响。
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Effect of in vivo ozone exposure to dorset sheep, an animal model with low levels of erythrocyte glucose-6-phosphate dehydrogenase activity.
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Ozone-induced decrease of mouse tracheal potential is not secondary to cellular inflammation.臭氧诱导的小鼠气管电位降低并非继发于细胞炎症。
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Animal models of human erythrocyte metabolic abnormalities.人类红细胞代谢异常的动物模型。
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本文引用的文献
1
A deficiency of glucose-6-phosphate dehydrogenase activity in erythrocytes from patients with favism.蚕豆病患者红细胞中葡萄糖-6-磷酸脱氢酶活性缺乏。
Bull Johns Hopkins Hosp. 1958 Apr;102(4):169-75.
2
The hemolytic effect of primaquine. VII. Biochemical studies of drug-sensitive erythrocytes.伯氨喹的溶血作用。VII. 药物敏感红细胞的生化研究。
J Lab Clin Med. 1955 Feb;45(2):286-95.
3
Double heterozygosity for glucose-6-phosphate dehydrogenase deficiency.葡萄糖-6-磷酸脱氢酶缺乏症的双重杂合性
J Med Genet. 1971 Jun;8(2):149-52. doi: 10.1136/jmg.8.2.149.
4
Genetic regulation of glucose 6-phosphate dehydrogenase activity in the inbred mouse.近交系小鼠中葡萄糖-6-磷酸脱氢酶活性的遗传调控
Biochem Genet. 1971 Aug;5(4):315-31. doi: 10.1007/BF00485859.
5
Ozone: a possible cause of hemolytic anemia in glucose-6-phosphate dehydrogenase deficient individuals.
J Toxicol Environ Health. 1977 Jan;2(3):709-12. doi: 10.1080/15287397709529470.
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