Szmolenszky T, Márk B, Kiss T, Trombitás K
Acta Chir Hung. 1984;25(2):107-18.
The fine structure of crural muscles in 11 patients suffering from post-thrombotic syndrome was investigated. The contractile elements and the mitochondria were mainly involved in the pathological changes. Glycogen granules in increased number and strong lipid vacuolization were shown. The focal occurrence of damages has to be emphasized. Beside, ultrastructural changes are induced by hypoxia and by other factors. It can be established that limited functional capacity of the lower extremities in post-thrombotic syndrome is due not only to chronic haemodynamic insufficiency but to the serious fine-structural damage.
对11例血栓形成后综合征患者的小腿肌肉精细结构进行了研究。收缩元件和线粒体主要参与了病理变化。糖原颗粒数量增加，出现了明显的脂质空泡化。必须强调损伤的局灶性发生。此外，超微结构变化是由缺氧和其他因素引起的。可以确定，血栓形成后综合征患者下肢有限的功能能力不仅归因于慢性血流动力学不足，还归因于严重的精细结构损伤。
