Estañol B, Lopez-Rios G
J Neurol Neurosurg Psychiatry. 1984 Aug;47(8):799-805. doi: 10.1136/jnnp.47.8.799.
When a patient with a peripheral monocular paresis is forced to look with the paretic eye, head movements induce the sensation of an unstable visual world. The patient behaves as if he had acute bilateral labyrinthine lesions. These symptoms are due to the lack of compensatory ocular movement and the patients complain that the visual objects move in the direction opposite to the head. The patients develop ataxia, nausea, vomiting and past pointing. The symptoms, however, are transient and consistently disappear after approximately 48 hours. The central adaptation to looking and seeing with the paralysed eye is associated with a plastic change of the VOR. This plastic adaptation is probably induced by the large retinal slip produced by the lack of compensatory movement of the eye and can be studied in the normally mobile eye in the dark. The psychophysical adaptation is probably generated by an efferent copy or corollary discharge of the vestibular system to the visual system that cancels the retinal error.
当患有外周单眼轻瘫的患者被迫用麻痹眼视物时，头部运动可诱发视觉世界不稳定的感觉。患者的表现就好像患有急性双侧迷路病变一样。这些症状是由于缺乏代偿性眼球运动所致，患者抱怨视觉对象朝着与头部相反的方向移动。患者会出现共济失调、恶心、呕吐和指物偏向。然而，这些症状是短暂的，大约48小时后会持续消失。中枢对用麻痹眼视物的适应与前庭眼反射（VOR）的可塑性变化有关。这种可塑性适应可能是由眼球缺乏代偿运动产生的大量视网膜滑移所诱发的，并且可以在黑暗中正常活动的眼睛中进行研究。心理物理学适应可能是由前庭系统向视觉系统发出的传出拷贝或伴随放电产生的，它消除了视网膜误差。
