[早期烧伤毒血症及暴露于中分子量血液肽期间线粒体的功能状态]
[Functional state of mitochondria during early burn toxemia and exposure to middle molecular weight blood peptides].
作者信息
Riabinin V E, Lifshits R I, Charnaia L F, Val'dman B M, Efimenko G P
出版信息
Vopr Med Khim. 1984 May-Jun;30(3):113-6.
PMID:6474934
Abstract
Accumulation of biologically active peptides, distinctly affecting the respiration and enzymatic activity of mitochondria, may be responsible for deterioration of oxidative phosphorylation occurring in mitochondria within the early period of burn toxemia.
摘要
生物活性肽的积累可能导致烧伤毒血症早期线粒体中发生的氧化磷酸化恶化，这种积累对线粒体的呼吸和酶活性有明显影响。
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