Degtiareva I I, Solodova E V
Ter Arkh. 1984;56(8):100-6.
It has been demonstrated that peptic ulcer patients manifest hypo- and dysaminoacidemia and a dramatic increase in the amino acid pool of the gastric mucosa which are accounted for by neurogenic dystrophy leading to catabolic orientation of the processes not only in the mucous membrane of the gastroduodenal zone but also in other organs with a high level of protein synthesis, particularly in the liver. Based on the coefficients that determine amino acid transformation in the liver and analysis of the blood serum content of replaceable amino acids, peptic ulcer patients were discovered to have marked impairment of the amino acid-forming function of the liver.
