Humes L E
J Acoust Soc Am. 1984 Nov;76(5):1318-29. doi: 10.1121/1.391447.
The interaction of noise with a variety of other agents and with some physical characteristics of the individual to produce noise-induced hearing loss is reviewed critically. The review is restricted, for the most part, to publications since 1970. Other agents interacting with steady-state noise that are reviewed here include: (1) ototoxic drugs (kanamycin, neomycin, ethacrynic acid, furosemide, and salicylates), (2) impulse noise, and (3) whole-body vibration. Physical characteristics of the individual that are reviewed are: (1) age, (2) presence of previous hearing loss from prior noise exposure, (3) eye color, and (4) race. Suggestions for future research in this general area are also made. Some of these suggestions are as follows: (1) to extend studies of the interaction of steady-state noise with impulse noise, salicylates, and whole-body vibration to encompass a broader range of exposure conditions, including exposure conditions typically encountered by the worker, (2) to develop an animal model of presbycusis to explore the interactions of noise-induced hearing loss and presbycusis, and (3) to explore the potential interactions resulting from concurrent exposure to multiple agents, such as impulse noise and ototoxic drugs, in younger, more susceptible animals.
本文对噪声与多种其他因素以及个体的某些身体特征相互作用导致噪声性听力损失的情况进行了批判性综述。该综述大部分限于1970年以来的出版物。本文所综述的与稳态噪声相互作用的其他因素包括：（1）耳毒性药物（卡那霉素、新霉素、依他尼酸、呋塞米和水杨酸盐），（2）脉冲噪声，以及（3）全身振动。所综述的个体身体特征包括：（1）年龄，（2）既往噪声暴露导致的听力损失情况，（3）眼睛颜色，以及（4）种族。还对该领域未来的研究提出了建议。其中一些建议如下：（1）将稳态噪声与脉冲噪声、水杨酸盐和全身振动相互作用的研究扩展到更广泛的暴露条件，包括工人通常遇到的暴露条件，（2）建立老年性聋动物模型，以探索噪声性听力损失与老年性聋的相互作用，以及（3）在更年轻、更易受影响的动物中探索同时暴露于多种因素（如脉冲噪声和耳毒性药物）所产生的潜在相互作用。
