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CNS hypomyelinated mutant mice (jimpy, shiverer, quaking): in vitro evidence for primary oligodendrocyte defects.

作者信息

Wolf M K, Billings-Gagliardi S

出版信息

Adv Exp Med Biol. 1984;181:115-33. doi: 10.1007/978-1-4684-4868-9_10.

DOI:10.1007/978-1-4684-4868-9_10
PMID:6532154
Abstract
摘要

相似文献

1
CNS hypomyelinated mutant mice (jimpy, shiverer, quaking): in vitro evidence for primary oligodendrocyte defects.
Adv Exp Med Biol. 1984;181:115-33. doi: 10.1007/978-1-4684-4868-9_10.
2
Oligodendrocyte abnormalities in shiverer mouse mutant are determined in primary chimaeras.
Nature. 1982 Sep 23;299(5881):357-9. doi: 10.1038/299357a0.
3
Radial component of the central myelin in neurologic mutant mice.神经突变小鼠中中央髓鞘的径向成分
Lab Invest. 1982 Jul;47(1):51-9.
4
Cultures of shiverer mutant cerebellum injected with normal oligodendrocytes make both normal and shiverer myelin.注射了正常少突胶质细胞的颤抖突变型小脑培养物可产生正常和颤抖型髓磷脂。
Proc Natl Acad Sci U S A. 1984 Apr;81(8):2558-61. doi: 10.1073/pnas.81.8.2558.
5
Order-disorder phenomena in myelinated nerve sheaths. VI. The effects of quaking, jimpy and shiverer mutations: an X-ray scattering study of mouse sciatic and optic nerves.有髓神经鞘中的有序-无序现象。VI. 颤抖、跳跃和颤抖者突变的影响：小鼠坐骨神经和视神经的X射线散射研究。
J Mol Biol. 1996 Feb 23;256(2):319-29. doi: 10.1006/jmbi.1996.0088.
6
The dysmyelinating mouse mutations shiverer (shi) and myelin deficient (shimld).脱髓鞘小鼠突变体颤抖鼠（shi）和髓磷脂缺陷鼠（shimld）。
Behav Genet. 1990 Mar;20(2):213-34. doi: 10.1007/BF01067791.
7
Quaking*jimpy double mutant mice: additional evidence for independence of primary deficits in jimpy.
Brain Res Mol Brain Res. 1990 Apr;7(3):189-98. doi: 10.1016/0169-328x(90)90027-b.
8
Absence of correlations between glutamine-synthetase activity and dysmyelination-associated modifications of astroglia in the brain of murine mutants.
Neurochem Res. 1986 Apr;11(4):527-33. doi: 10.1007/BF00965322.
9
Sodium channel density in hypomyelinated brain increased by myelin basic protein gene deletion.髓鞘碱性蛋白基因缺失导致低髓鞘化脑内钠通道密度增加。
Nature. 1991 Aug 1;352(6334):431-4. doi: 10.1038/352431a0.
10
Glial hyaluronate-binding protein in dysmyelinating mice mutants: jimpy, quaking and shiverer.
Acta Neuropathol. 1989;79(3):326-9. doi: 10.1007/BF00294669.

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Cells. 2022 Sep 30;11(19):3085. doi: 10.3390/cells11193085.
2
Quaking but not parkin is the major tumor suppressor in 6q deleted region in glioblastoma.在胶质母细胞瘤6q缺失区域，震颤蛋白而非帕金蛋白是主要的肿瘤抑制因子。
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3
Myelin Basic Protein Cleaves Cell Adhesion Molecule L1 and Improves Regeneration After Injury.
髓鞘碱性蛋白切割细胞粘附分子L1并改善损伤后的再生。
Mol Neurobiol. 2016 Jul;53(5):3360-3376. doi: 10.1007/s12035-015-9277-0. Epub 2015 Jun 17.
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Myelin basic protein cleaves cell adhesion molecule L1 and promotes neuritogenesis and cell survival.髓鞘碱性蛋白裂解细胞黏附分子 L1 并促进神经突生成和细胞存活。
J Biol Chem. 2014 May 9;289(19):13503-18. doi: 10.1074/jbc.M113.530238. Epub 2014 Mar 26.
5
Region-specific myelin pathology in mice lacking the golli products of the myelin basic protein gene.缺乏髓鞘碱性蛋白基因的golli产物的小鼠的区域特异性髓鞘病理
J Neurosci. 2005 Jul 27;25(30):7004-13. doi: 10.1523/JNEUROSCI.0288-05.2005.
6
The pathobiology of myelin mutants reveal novel biological functions of the MBP and PLP genes.髓磷脂突变体的病理生物学揭示了髓鞘碱性蛋白（MBP）和髓磷脂蛋白脂蛋白（PLP）基因的新生物学功能。
Brain Pathol. 2001 Jan;11(1):74-91. doi: 10.1111/j.1750-3639.2001.tb00383.x.