Sylvén C, Berglund B, Kaijser L
Clin Physiol. 1984 Jun;4(3):221-6. doi: 10.1111/j.1475-097x.1984.tb00116.x.
Thirteen patients with severe stable angina pectoris were studied by coronary sinus catheterization. In all patients, severe chest pains were produced by atrial pacing. The chest pains had disappeared within 10 min after pacing. Simultaneous arterial (a) and coronary sinus (cs) blood samples were taken before, during and after pacing and analyzed for myoglobin and lactate. The a-cs difference of myoglobin tended to become more negative after pacing, although the change was not significant. However, the change in negative direction post-pacing of the a-cs myoglobin difference was quantitatively correlated with the change in negative direction of the a-cs lactate difference during pacing in the individual patients. This suggests that short-term myocardial ischaemia without signs of established myocardial infarction may provoke myocardial myoglobin release.
对13例严重稳定型心绞痛患者进行了冠状窦导管插入术研究。在所有患者中，心房起搏均引发了严重胸痛。胸痛在起搏后10分钟内消失。在起搏前、起搏期间和起搏后同时采集动脉（a）血样和冠状窦（cs）血样，并分析其中的肌红蛋白和乳酸。起搏后肌红蛋白的a-cs差值趋于更负，尽管变化不显著。然而，个体患者起搏后a-cs肌红蛋白差值负向变化在数量上与起搏期间a-cs乳酸差值负向变化相关。这表明无已确诊心肌梗死迹象的短期心肌缺血可能会引发心肌肌红蛋白释放。
