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[同时性双侧高血压性颅内血肿]

[Simultaneous, bilateral hypertensive intracranial hematomas].

作者信息

Tanikake T, Kawaguchi S, Tada T, Kyoi K, Utsumi S, Nakasone K

出版信息

No Shinkei Geka. 1983 Oct;11(10):1085-90.

PMID:6646340
Abstract

The reported incidences of bilateral intracerebral hemorrhages due to systemic arterial hypertension are exceptionally rare in Japan. Unilateral hemorrhages, on the other hand, are less uncommon. Recently, we have examined two patients with bilateral intracerebral hemorrhages due to hypertension. The first case involved bilateral thalamic hemorrhages; and in the other, a contralateral hemorrhage developed postoperatively, subsequent to the evacuation of a primary hematoma. The characteristic neurological manifestation of bilateral intracerebral hemorrhages include quadriparesis, bilateral Babinski's signs, stupor, and coma. Published information regarding the anatomy of intracerebral hemorrhages due to hypertension is inconclusive, but the bilateral basal ganglias are believed to be most frequently involved. One school of thought explains the pathomechanism of bilateral hemorrhages as a symmetrical rupture of cerebral microaneurysm. However, it is possible that an unilateral hematoma was formed by a ruptured microaneurysm, and subsequently, a contralateral hemorrhage developed in relatively short time due to circulatory disturbance. As in the case of general cerebral hemorrhage, a craniotomy is also indicated for hypertensive bilateral intracerebral hemorrhage.

摘要

据报道,在日本,由系统性动脉高血压引起的双侧脑出血发病率极为罕见。另一方面,单侧出血则较为常见。最近,我们诊治了两名因高血压导致双侧脑出血的患者。第一例为双侧丘脑出血;另一例则是在原发性血肿清除术后出现了对侧出血。双侧脑出血的典型神经学表现包括四肢瘫痪、双侧巴宾斯基征、昏睡和昏迷。关于高血压性脑出血的解剖学已发表的信息尚无定论,但双侧基底节被认为是最常受累的部位。一种观点认为双侧出血的发病机制是脑微动脉瘤的对称性破裂。然而,也有可能是微动脉瘤破裂形成了单侧血肿,随后由于循环障碍在相对较短的时间内发生了对侧出血。与一般脑出血的情况一样,高血压性双侧脑出血也需要进行开颅手术。

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