Fricker H S, Sauter M, Buchs B
Z Geburtshilfe Perinatol. 1983 Jan-Feb;187(1):50-3.
A para III was found to have a constantly silent CTG. In two fetal blood analyses the pH was normal. During the first few hours post partum the infant had rapidly increasing neurologic disturbances with violent convulsions and coma. As early as on the first day of life the computer tomogram showed extensive, later persistent hypodense zones corresponding to severe asphyxial cerebral necrosis. Based an the course of CT changes it has to be assumed that the hypoxic crisis occurred some days prior to the onset of labor. Pathologic changes in the umbilical cord indicated that the cause could have been a transitory occlusion in utero. The computer tomogram enables cerebral insults to be dated more accurately. If prenatal hypoxia occurs repeatedly new methods of prevention must be sought.
发现一名III级患者的CTG持续无变化。两次胎儿血液分析显示pH值正常。产后最初几个小时内，婴儿的神经功能障碍迅速加重，出现剧烈惊厥和昏迷。早在出生第一天，计算机断层扫描就显示出广泛的、后来持续存在的低密度区，对应于严重的窒息性脑坏死。根据CT变化过程，推测缺氧危机发生在分娩开始前几天。脐带的病理变化表明，病因可能是子宫内的短暂闭塞。计算机断层扫描能够更准确地确定脑损伤的时间。如果反复发生产前缺氧，必须寻求新的预防方法。
