Kenning J A, Heros R C, Dujovny M, Latchaw R E, Nelson D
Surg Neurol. 1984 Feb;21(2):159-64. doi: 10.1016/0090-3019(84)90335-5.
The incidence and severity of cerebral vasospasm and hydrocephalus following induced subarachnoid hemorrhage in an experimental group of animals that subsequently received epsilon-aminocaproic acid was compared to that seen in a control group that received no antifibrinolytic therapy. No augmentation of either vasospasm or hydrocephalus could be attributed to the epsilon-aminocaproic acid in the treated as compared to the control group.
将随后接受ε-氨基己酸治疗的实验组动物诱导性蛛网膜下腔出血后脑血管痉挛和脑积水的发生率及严重程度，与未接受抗纤维蛋白溶解治疗的对照组进行比较。与对照组相比，治疗组中无论是脑血管痉挛还是脑积水，均未发现可归因于ε-氨基己酸的增加情况。
