异常纤维蛋白溶解：原发性淋巴水肿患者脂性皮肤硬化症或“慢性蜂窝织炎”的病因。

Abnormal fibrinolysis: the cause of lipodermatosclerosis or "chronic cellulitis" in patients with primary lymphedema.

作者信息

Stewart G, Pattison M, Burnand K G

出版信息

Lymphology. 1984 Mar;17(1):23-7.

Abstract

Blood fibrinolytic activity was measured in 20 patients with primary lymphedema, ten without and ten with skin changes usually attributed to "chronic cellulitis". The patients with abnormal skin showed reduced fibrinolytic activity, a finding previously described in patients with chronic venous disease and lipodermatosclerosis . It is postulated that changes of "chronic cellulitis" are identical to lipodermatosclerosis and are produced by a similar mechanism, namely reduced fibrinolysis.

摘要

对20例原发性淋巴水肿患者进行了血液纤溶活性测定，其中10例无皮肤改变，10例有通常归因于“慢性蜂窝织炎”的皮肤改变。皮肤异常的患者纤溶活性降低，这一发现先前在慢性静脉疾病和脂肪皮肤硬化症患者中已有描述。据推测，“慢性蜂窝织炎”的改变与脂肪皮肤硬化症相同，且由类似机制产生，即纤溶作用降低。