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Spontaneous T-cell lymphokine production and enhanced macrophage Ia expression and tumoricidal activity in MRL-lpr mice.

作者信息

Lu C Y, Unanue E R

出版信息

Clin Immunol Immunopathol. 1982 Nov;25(2):213-22. doi: 10.1016/0090-1229(82)90184-2.

DOI:10.1016/0090-1229(82)90184-2
PMID:6762274
Abstract
摘要

相似文献

1
Spontaneous T-cell lymphokine production and enhanced macrophage Ia expression and tumoricidal activity in MRL-lpr mice.MRL-lpr小鼠中T细胞自发产生淋巴因子以及巨噬细胞Ia表达增强和杀肿瘤活性增强
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2
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Cell Immunol. 1984 Aug;87(1):92-100. doi: 10.1016/0008-8749(84)90133-3.
3
Functional analysis of macrophage hybridomas. II. Antibody-independent tumor cytotoxicity and its dissociation from IL-1 production and Ia expression.巨噬细胞杂交瘤的功能分析。II. 不依赖抗体的肿瘤细胞毒性及其与白细胞介素-1产生和Ia表达的分离
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4
Dysfunction of Ia-positive antigen-presenting cells in autoimmune mice.
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5
Growth regulation of transformed T cells by nonactivated macrophages: the role of Ia expression.未活化巨噬细胞对转化T细胞的生长调节：Ia表达的作用。
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6
Autoreactive T cells in MRL/Mpr-lpr/lpr mice. Characterization of the lymphokines produced and analysis of antigen-presenting cells required.MRL/Mpr-lpr/lpr小鼠中的自身反应性T细胞。所产生淋巴因子的特性及所需抗原呈递细胞的分析。
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7
Spleen cell factor inhibits lymphoproliferation, abnormal Ia expression and overt autoimmunity in MRL-lpr mice.脾细胞因子可抑制MRL-lpr小鼠的淋巴细胞增殖、异常Ia表达及明显的自身免疫反应。
Clin Immunol Immunopathol. 1986 Oct;41(1):145-53. doi: 10.1016/0090-1229(86)90058-9.
8
Spontaneous production of interleukin 3 by T lymphocytes from autoimmune MRL/MP-lpr/lpr mice.来自自身免疫性MRL/MP-lpr/lpr小鼠的T淋巴细胞自发产生白细胞介素3 。
Eur J Immunol. 1984 Jul;14(7):599-605. doi: 10.1002/eji.1830140704.
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Macrophage activation by interferon: dissociation between tumoricidal capacity and suppressive activity.干扰素对巨噬细胞的激活作用：杀瘤能力与抑制活性之间的分离
Eur J Immunol. 1982 Apr;12(4):320-6. doi: 10.1002/eji.1830120412.
10
Macrophages as effector cells of protective immunity in murine schistosomiasis. VI. T cell-dependent, lymphokine-mediated, activation of macrophages in response to Schistosoma mansoni antigens.巨噬细胞作为小鼠血吸虫病保护性免疫的效应细胞。VI. 针对曼氏血吸虫抗原，T细胞依赖性、淋巴因子介导的巨噬细胞激活。
J Immunol. 1983 Sep;131(3):1481-6.

引用本文的文献

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Interferon-gamma is required for lupus-like disease and lymphoaccumulation in MRL-lpr mice.MRL-lpr小鼠出现狼疮样疾病和淋巴细胞积聚需要γ干扰素。
J Clin Invest. 1998 Jan 15;101(2):364-71. doi: 10.1172/JCI750.
2
Immune complex-degradation ability of macrophages in MRL/Mp-lpr/lpr lupus mice and its regulation by cytokines.MRL/Mp-lpr/lpr狼疮小鼠巨噬细胞的免疫复合物降解能力及其细胞因子调控
Clin Exp Immunol. 1994 Jan;95(1):115-21. doi: 10.1111/j.1365-2249.1994.tb06024.x.
3
The role of cytokines in the immunopathogenesis of lupus.细胞因子在狼疮免疫发病机制中的作用。
Springer Semin Immunopathol. 1994;16(2-3):153-80. doi: 10.1007/BF00197515.
4
The role of nitric oxide in the pathogenesis of spontaneous murine autoimmune disease: increased nitric oxide production and nitric oxide synthase expression in MRL-lpr/lpr mice, and reduction of spontaneous glomerulonephritis and arthritis by orally administered NG-monomethyl-L-arginine.一氧化氮在自发性小鼠自身免疫性疾病发病机制中的作用：MRL-lpr/lpr小鼠中一氧化氮生成增加及一氧化氮合酶表达上调，口服NG-单甲基-L-精氨酸可减轻自发性肾小球肾炎和关节炎
J Exp Med. 1994 Feb 1;179(2):651-60. doi: 10.1084/jem.179.2.651.
5
Increased Ia expression, T lymphocyte subset abnormalities and autoimmunity in murine strains bearing the lpr gene.携带lpr基因的小鼠品系中Ia表达增加、T淋巴细胞亚群异常与自身免疫
Clin Exp Immunol. 1984 Oct;58(1):145-53.
6
Macrophage-directed lymphokines.巨噬细胞定向淋巴因子
Surv Immunol Res. 1984;3(2-3):154-60. doi: 10.1007/BF02918783.
7
Regulation of human peripheral blood monocyte DR antigen expression in vitro by lymphokines and recombinant interferons.细胞因子和重组干扰素对人外周血单核细胞DR抗原体外表达的调控
J Clin Invest. 1984 Feb;73(2):556-65. doi: 10.1172/JCI111243.
8
Association of lpr gene with graft-vs.-host disease-like syndrome.lpr基因与移植物抗宿主病样综合征的关联。
J Exp Med. 1985 Jul 1;162(1):1-18. doi: 10.1084/jem.162.1.1.
9
Serum amyloid P component and autoimmune parameters in the assessment of arthritis activity in MRL/lpr/lpr mice.血清淀粉样蛋白P成分和自身免疫参数在评估MRL/lpr/lpr小鼠关节炎活动中的作用
Clin Exp Immunol. 1985 Sep;61(3):509-16.
10
Relationship of macrophages to defective delayed-type hypersensitivity in B6/lpr mice.B6/lpr小鼠中巨噬细胞与缺陷性迟发型超敏反应的关系。
Clin Exp Immunol. 1986 Dec;66(3):599-605.