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Cholera toxin activation of adenylate cyclase. Roles of nucleoside triphosphates and a macromolecular factor in the ADP ribosylation of the GTP-dependent regulatory component.

作者信息

Enomoto K, Gill D M

出版信息

J Biol Chem. 1980 Feb 25;255(4):1252-8.

PMID:6766444
Abstract
摘要

相似文献

1
Cholera toxin activation of adenylate cyclase. Roles of nucleoside triphosphates and a macromolecular factor in the ADP ribosylation of the GTP-dependent regulatory component.霍乱毒素对腺苷酸环化酶的激活作用。核苷三磷酸和一种大分子因子在依赖鸟苷三磷酸的调节成分的ADP核糖基化中的作用。
J Biol Chem. 1980 Feb 25;255(4):1252-8.
2
A second guanyl nucleotide-binding site associated with adenylate cyclase. Distinct nucleotides activate adenylate cyclase and permit ADP-ribosylation by cholera toxin.与腺苷酸环化酶相关的第二个鸟苷酸结合位点。不同的核苷酸激活腺苷酸环化酶并允许霍乱毒素进行ADP-核糖基化。
J Biol Chem. 1983 Oct 10;258(19):11908-14.
3
ADP-ribosylation of membrane proteins and activation of adenylate cyclase by cholera toxin in fat cell ghosts from euthyroid and hypothyroid rats.来自甲状腺功能正常和甲状腺功能减退大鼠的脂肪细胞膜蛋白的ADP-核糖基化及霍乱毒素对腺苷酸环化酶的激活作用
Biochim Biophys Acta. 1979 Sep 3;586(3):518-27. doi: 10.1016/0304-4165(79)90042-4.
4
Requirements for cholera toxin-dependent ADP-ribosylation of the purified regulatory component of adenylate cyclase.腺苷酸环化酶纯化调节成分的霍乱毒素依赖性ADP-核糖基化的要求。
J Biol Chem. 1982 Jan 10;257(1):20-3.
5
The sites on the regulatory component of adenylate cyclase which are ADP-ribosylated by cholera toxin.
Biochem Biophys Res Commun. 1982 Apr 14;105(3):928-34. doi: 10.1016/0006-291x(82)91059-2.
6
Cholera toxin action on rabbit corpus luteum membranes: effects on adenylyl cyclase activity and adenosine diphospho-ribosylation of the stimulatory guanine nucleotide-binding regulatory component.霍乱毒素对兔黄体细胞膜的作用:对腺苷酸环化酶活性及刺激性鸟嘌呤核苷酸结合调节成分的腺苷二磷酸核糖基化作用的影响
Biol Reprod. 1985 Mar;32(2):463-74. doi: 10.1095/biolreprod32.2.463.
7
Mn2+-uncoupling of the catecholamine-sensitive adenylate cyclase system of rat reticulocytes. Parallel effects on cholera toxin-catalyzed ADP-ribosylation of the system.大鼠网织红细胞儿茶酚胺敏感的腺苷酸环化酶系统的锰离子解偶联。对霍乱毒素催化的该系统ADP核糖基化的平行作用。
Biochim Biophys Acta. 1981 Nov 5;677(3-4):408-16. doi: 10.1016/0304-4165(81)90254-3.
8
The role of guanine nucleotides in regulation of adenylate cyclase activity.鸟嘌呤核苷酸在腺苷酸环化酶活性调节中的作用。
Recent Prog Horm Res. 1981;37:635-65. doi: 10.1016/b978-0-12-571137-1.50018-9.
9
GTP stabilization of adenylate cyclase activated and ADP-ribosylated by choleragen.霍乱毒素激活并进行 ADP 核糖基化修饰的腺苷酸环化酶的 GTP 稳定作用。
Biochem Biophys Res Commun. 1981 Sep 16;102(1):66-74. doi: 10.1016/0006-291x(81)91489-3.
10
Demonstration of choleragen-dependent ADP-ribosylation in whole cells and correlation with the activation of adenylate cyclase.
Life Sci. 1980 Apr 28;26(17):1385-96. doi: 10.1016/0024-3205(80)90041-7.

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mBio. 2019 Oct 15;10(5):e02398-19. doi: 10.1128/mBio.02398-19.
2
Regulating the large Sec7 ARF guanine nucleotide exchange factors: the when, where and how of activation.调控大型 Sec7 ARF 鸟嘌呤核苷酸交换因子:激活的时机、地点和方式。
Cell Mol Life Sci. 2014 Sep;71(18):3419-38. doi: 10.1007/s00018-014-1602-7. Epub 2014 Apr 13.
3
Ras history: The saga continues.
Ras的历程:传奇仍在继续。
Small GTPases. 2010 Jul;1(1):2-27. doi: 10.4161/sgtp.1.1.12178.
4
The regulatory RAB and ARF GTPases for vesicular trafficking.用于囊泡运输的调节性RAB和ARF GTP酶。
Plant Physiol. 2008 Aug;147(4):1516-26. doi: 10.1104/pp.108.121798.
5
Fluid secretion caused by aerolysin-like hemolysin of Aeromonas sobria in the intestines is due to stimulation of production of prostaglandin E2 via cyclooxygenase 2 by intestinal cells.温和气单胞菌的气溶素样溶血素引起的肠道液体分泌是由于肠道细胞通过环氧化酶2刺激前列腺素E2的产生。
Infect Immun. 2008 Mar;76(3):1076-82. doi: 10.1128/IAI.01098-07. Epub 2007 Dec 17.
6
Nomenclature for the human Arf family of GTP-binding proteins: ARF, ARL, and SAR proteins.人源GTP结合蛋白Arf家族的命名法:ARF、ARL和SAR蛋白。
J Cell Biol. 2006 Feb 27;172(5):645-50. doi: 10.1083/jcb.200512057.
7
Selection of a WEHI-3B leukemia cell subclone resistant to inhibition by cholera toxin.选择对霍乱毒素抑制具有抗性的WEHI-3B白血病细胞亚克隆。
Mol Cell Biochem. 2002 Apr;233(1-2):19-26. doi: 10.1023/a:1015593226414.
8
Case studies in cholera: lessons in medical history and science.霍乱病例研究:医学史与科学中的经验教训
Yale J Biol Med. 1999 Nov-Dec;72(6):393-408.
9
The different inhibiting effect of cholera toxin on two leukemia cell lines does not correlate with their toxin binding capacity.霍乱毒素对两种白血病细胞系的不同抑制作用与其毒素结合能力无关。
Mol Cell Biochem. 1995 Nov 22;152(2):103-12. doi: 10.1007/BF01076072.
10
ADP-ribosylation factors: a family of approximately 20-kDa guanine nucleotide-binding proteins that activate cholera toxin.ADP核糖基化因子:一类分子量约为20 kDa的鸟嘌呤核苷酸结合蛋白家族,可激活霍乱毒素。
Mol Cell Biochem. 1994 Sep;138(1-2):157-66. doi: 10.1007/BF00928458.