Futagawa S, Fukazawa M, Horisawa M, Musha H, Ito T, Sugiura M, Kameda H, Okuda K
AJR Am J Roentgenol. 1980 May;134(5):917-23. doi: 10.2214/ajr.134.5.917.
Intraoperative and transhepatic portograms in patients with noncirrhotic portal hypertension, or idiopathic portal hypertension, were compared with portograms of patients with liver cirrhosis. Although the portograms in idiopathic portal hypertension varied from case to case, they were distinctly different from those in cirrhosis. The most common features of the former included a paucity of medium-sized portal branches, irregular and often obtuse-angled division of the peripheral branches, occasional abrupt interruptions of them, an avascular area beneath the liver surface, nonopacification of some of the large intrahepatic portal branches and of their periphery, and increase of very fine vasculature around large intrahepatic portal branches. These findings are compatible with occlusion of intrahepatic portal vessels at various levels.
对非肝硬化门静脉高压症（即特发性门静脉高压症）患者的术中及经肝门静脉造影与肝硬化患者的门静脉造影进行了比较。尽管特发性门静脉高压症患者的门静脉造影因病例而异，但与肝硬化患者的门静脉造影明显不同。前者最常见的特征包括中等大小门静脉分支稀少、外周分支不规则且常呈钝角分支、偶尔出现突然中断、肝表面下方无血管区、部分肝内大门静脉分支及其周边未显影，以及肝内大门静脉分支周围极细血管增多。这些发现与肝内各级门静脉血管闭塞相符。
