Tissot Guerraz F T, Sepetjan M, Monier J C
Ann Immunol (Paris). 1980 Nov-Dec;131D(3):289-98.
This adoptive transfer was studied in an inbred strain of syrian hamsters. Spleen and lymph node lymphocytes, from TFB-infected hamsters, were injected to isogeneic recipients. Anti-TFB immunity was studied in the recipients either by looking for the apparition of cutaneous lesions after superinfection by TFB, or by counting of Listeria monocytogenes remaining in the liver and in the spleen 48 h after a mixture of TFB and L. monocytogenes had been inoculated. The first method indicated that a complete inhibition of cutaneous lesions was obtained only with hyperimmunized donors. Our results also indicate that anti-TFB immunity was not medicated by B lymphocytes but by sensitized T lymphocytes.
在叙利亚仓鼠的一个近交系中研究了这种过继转移。将来自感染TFB的仓鼠的脾脏和淋巴结淋巴细胞注射给同基因受体。通过观察TFB再次感染后皮肤损伤的出现情况，或者通过在接种TFB和单核细胞增生李斯特菌混合物48小时后计数肝脏和脾脏中残留的单核细胞增生李斯特菌，来研究受体中的抗TFB免疫。第一种方法表明，只有用超免疫供体才能完全抑制皮肤损伤。我们的结果还表明，抗TFB免疫不是由B淋巴细胞介导的，而是由致敏T淋巴细胞介导的。
