Povzhitkov M M, Sagach V F, Shablovskaya O V
Cor Vasa. 1978;20(1):63-71.
In the course of experimental cardiogenic shock in dogs it was found that in comparison with marked disturbances of heart function and haemodynamics in the systemic circulation, the changes in the right ventricular contractility were less conspicuous. The pulmonary arterial pressure kept at a level approaching the initial one. The pulmonary vascular resistance substantially rose at the same time, especially so in the arterioles. The left heart filling pressure did not increase. The authors assume that the changes in the pulmonary vascular resistance result from direct neurohumoral control of vascular resistance, and discuss possible mechanism of this control.
在犬实验性心源性休克过程中发现，与体循环中心脏功能和血流动力学的明显紊乱相比，右心室收缩性的变化不太明显。肺动脉压维持在接近初始水平。与此同时，肺血管阻力显著升高，尤其是在小动脉中。左心充盈压并未升高。作者认为肺血管阻力的变化是由血管阻力的直接神经体液控制引起的，并讨论了这种控制的可能机制。
