Criado A, Gomez-Arnau J, Luengo C, Gilsanz F, Dominguez E, Avello F
Resuscitation. 1981 Dec;9(4):353-7. doi: 10.1016/0300-9572(81)90011-3.
We present a case of acute lithium ion intoxication in which the main clinical feature was respiratory failure secondary to pulmonary oedema possibly due to myocardial depression, associated with profound stupor. The initial lithium ion concentrations were 3.15 mmol/l in plasma and 27.6 mmol/l in urine. Under symptomatic treatment with oxygen, digitalis and diuretics, both the pulmonary and the neurological disorders reverted gradually, in parallel with the decreasing plasma lithium ion concentrations.
我们报告一例急性锂离子中毒病例，其主要临床特征为继发于肺水肿的呼吸衰竭，肺水肿可能是由于心肌抑制所致，并伴有深度昏迷。血浆中初始锂离子浓度为3.15 mmol/L，尿液中为27.6 mmol/L。在给予吸氧、洋地黄和利尿剂的对症治疗下，随着血浆锂离子浓度的降低，肺部和神经系统疾病均逐渐好转。
