实验性自身免疫性甲状腺炎的调控：小鼠H-2 I-A亚区易感性图谱 - Suppr

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超能文献

Regulation of experimental autoimmune thyroiditis: mapping of susceptibility to the I-A subregion of the mouse H-2.

作者信息

Beisel K W, David C S, Giraldo A A, Kong Y M, Rose N R

出版信息

Immunogenetics. 1982;15(4):427-30. doi: 10.1007/BF00364266.

DOI:10.1007/BF00364266

PMID:6804377

Abstract

摘要

相似文献

Regulation of experimental autoimmune thyroiditis: mapping of susceptibility to the I-A subregion of the mouse H-2.实验性自身免疫性甲状腺炎的调控：小鼠H-2 I-A亚区易感性图谱

Immunogenetics. 1982;15(4):427-30. doi: 10.1007/BF00364266.

Regulation of experimental autoimmune thyroiditis: influence of non-H-2 genes.实验性自身免疫性甲状腺炎的调控：非H-2基因的影响

J Immunogenet. 1982 Aug;9(4):257-65. doi: 10.1111/j.1744-313x.1982.tb00981.x.

Mutations at H-2K or H-2D alter immune response phenotype of autoimmune thyroiditis.H-2K或H-2D处的突变会改变自身免疫性甲状腺炎的免疫反应表型。

Immunogenetics. 1982;15(6):625-7. doi: 10.1007/BF00347059.

H-2K mutation controls immune response phenotype of autoimmune thyroiditis. Critical expression of mutant gene product in both thymus and thyroid glands.H-2K突变控制自身免疫性甲状腺炎的免疫反应表型。突变基因产物在胸腺和甲状腺中均有关键表达。

J Exp Med. 1980 Oct 1;152(4):1115-20. doi: 10.1084/jem.152.4.1115.

Gene(s) at I-A subregion controls the autoimmune response to thyroglobulin in mice.位于I-A亚区的基因控制小鼠对甲状腺球蛋白的自身免疫反应。

Immunol Commun. 1981;10(4-5):341-7. doi: 10.3109/08820138109050699.

The genetics of thyroiditis as a prototype of human autoimmune disease.作为人类自身免疫性疾病原型的甲状腺炎遗传学

Ann Allergy. 1985 Apr;54(4):261-71.

Mutation in the IA subregion of the murine H-2 complex.小鼠H-2复合体IA亚区的突变

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MHC class I polymorphism in lupus-prone MRL/Mp mice.狼疮易感MRL/Mp小鼠中的主要组织相容性复合体I类多态性

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Genetic control of experimental autoimmune myasthenia gravis in mice. I. Lymphocyte proliferative response to acetylcholine receptors is under H-2-linked Ir gene control.小鼠实验性自身免疫性重症肌无力的遗传控制。I. 对乙酰胆碱受体的淋巴细胞增殖反应受H-2连锁免疫反应基因控制。

J Immunol. 1979 Dec;123(6):2540-3.

Mutation at H-2K locus influences susceptibility to autoimmune thyroiditis.H-2K基因座的突变会影响自身免疫性甲状腺炎的易感性。

Nature. 1979 Jun 21;279(5715):715-6. doi: 10.1038/279715a0.

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Genes Outside the Major Histocompatibility Complex Locus Are Linked to the Development of Thyroid Autoantibodies and Thyroiditis in NOD.H2h4 Mice.主要组织相容性复合体基因座以外的基因与NOD.H2h4小鼠甲状腺自身抗体的产生及甲状腺炎的发生相关。

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Evidence that MHC I-E dampens thyroid autoantibodies and prevents spreading to a second thyroid autoantigen in I-A(k) NOD mice.有证据表明，在I-A(k) NOD小鼠中，MHC I-E可抑制甲状腺自身抗体，并防止扩散至第二种甲状腺自身抗原。

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Efficacy of HLA-DRB1∗03:01 and H2E transgenic mouse strains to correlate pathogenic thyroglobulin epitopes for autoimmune thyroiditis.HLA-DRB1∗03:01 和 H2E 转基因小鼠品系对自身免疫性甲状腺炎相关致病性甲状腺球蛋白表位的功效。

J Autoimmun. 2011 Sep;37(2):63-70. doi: 10.1016/j.jaut.2011.05.010. Epub 2011 Jun 17.

The genetics of autoimmune thyroiditis: the first decade.自身免疫性甲状腺炎的遗传学：第一个十年。

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cDNA immunization of mice with human thyroglobulin generates both humoral and T cell responses: a novel model of thyroid autoimmunity.用人类甲状腺球蛋白对小鼠进行 cDNA 免疫接种可产生体液和 T 细胞应答：一种新型甲状腺自身免疫模型。

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Autoimmune thyroiditis: a model uniquely suited to probe regulatory T cell function.自身免疫性甲状腺炎：一种特别适合探究调节性 T 细胞功能的模型。

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'Linkage analysis of thyroid antibody production: evidence for shared susceptibility to clinical autoimmune thyroid disease.甲状腺抗体产生的连锁分析：临床自身免疫性甲状腺疾病共同易感性的证据。

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A novel H2A-E+ transgenic model susceptible to human but not mouse thyroglobulin-induced autoimmune thyroiditis: identification of mouse pathogenic epitopes.一种对人甲状腺球蛋白而非小鼠甲状腺球蛋白诱导的自身免疫性甲状腺炎易感的新型H2A-E +转基因模型：小鼠致病表位的鉴定

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H2E-derived Ealpha52-68 peptide presented by H2Ab interferes with clonal deletion of autoreactive T cells in autoimmune thyroiditis.由H2Ab呈递的源自H2E的Ealpha52 - 68肽干扰自身免疫性甲状腺炎中自身反应性T细胞的克隆清除。

J Immunol. 2008 May 15;180(10):7039-46. doi: 10.4049/jimmunol.180.10.7039.

Thyroglobulin as an autoantigen: what can we learn about immunopathogenicity from the correlation of antigenic properties with protein structure?甲状腺球蛋白作为自身抗原：从抗原特性与蛋白质结构的相关性中我们能了解到哪些关于免疫致病性的信息？

Immunology. 2004 May;112(1):13-25. doi: 10.1111/j.1365-2567.2004.01861.x.

本文引用的文献

T-cell regulation in autoimmune thyroiditis.自身免疫性甲状腺炎中的T细胞调节

Immunol Rev. 1981;55:299-314. doi: 10.1111/j.1600-065x.1981.tb00346.x.

Autoimmune murine thyroiditis relation to histocompatibility (H-2) type.自身免疫性小鼠甲状腺炎与组织相容性（H-2）类型的关系。

Science. 1971 Dec 10;174(4014):1137-9. doi: 10.1126/science.174.4014.1137.

Murine thyroiditis: importance of adjuvant and mouse strain for the induction of thyroid lesions.小鼠甲状腺炎：佐剂和小鼠品系在诱导甲状腺病变中的重要性。

J Immunol. 1971 Mar;106(3):698-704.

Autoimmune murine thyroiditis. IV. Localization of genetic control of the immune response.自身免疫性小鼠甲状腺炎。IV. 免疫反应遗传控制的定位

J Immunol. 1974 Mar;112(3):965-9.

Aberrant thymus tissue in rat and mouse thyroid.

Experientia. 1972 Jan 15;28(1):79-81. doi: 10.1007/BF01928275.

Mutation at H-2K locus influences susceptibility to autoimmune thyroiditis.H-2K基因座的突变会影响自身免疫性甲状腺炎的易感性。

Nature. 1979 Jun 21;279(5715):715-6. doi: 10.1038/279715a0.

Two new recombinant H-2 haplotypes, one of which juxtaposes Kb and Ik alleles.

J Immunol. 1979 Nov;123(5):1965-9.

Regulation of autoimmune response to mouse thyroglobulin: influence of H-2D-end genes.对小鼠甲状腺球蛋白自身免疫反应的调节：H-2D末端基因的影响。

J Immunol. 1979 Jul;123(1):15-8.

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