Mutation at H-2K locus influences susceptibility to autoimmune thyroiditis.
作者信息
Maron R, Cohen I R
出版信息
Nature. 1979 Jun 21;279(5715):715-6. doi: 10.1038/279715a0.
DOI:10.1038/279715a0
PMID:450120
Abstract
摘要
相似文献
1
Mutation at H-2K locus influences susceptibility to autoimmune thyroiditis.H-2K基因座的突变会影响自身免疫性甲状腺炎的易感性。
Nature. 1979 Jun 21;279(5715):715-6. doi: 10.1038/279715a0.
2
H-2K mutation controls immune response phenotype of autoimmune thyroiditis. Critical expression of mutant gene product in both thymus and thyroid glands.H-2K突变控制自身免疫性甲状腺炎的免疫反应表型。突变基因产物在胸腺和甲状腺中均有关键表达。
J Exp Med. 1980 Oct 1;152(4):1115-20. doi: 10.1084/jem.152.4.1115.
3
Mutations at H-2K or H-2D alter immune response phenotype of autoimmune thyroiditis.H-2K或H-2D处的突变会改变自身免疫性甲状腺炎的免疫反应表型。
Immunogenetics. 1982;15(6):625-7. doi: 10.1007/BF00347059.
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Gene(s) at I-A subregion controls the autoimmune response to thyroglobulin in mice.位于I-A亚区的基因控制小鼠对甲状腺球蛋白的自身免疫反应。
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Regulation of experimental autoimmune thyroiditis: influence of non-H-2 genes.实验性自身免疫性甲状腺炎的调控：非H-2基因的影响
J Immunogenet. 1982 Aug;9(4):257-65. doi: 10.1111/j.1744-313x.1982.tb00981.x.
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Thymic reconstitution of H-2-linked T-cell responses to thyroglobulin or insulin.H-2连锁的针对甲状腺球蛋白或胰岛素的T细胞反应的胸腺重建。
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Relation between the major histocompatibility (B) locus and autoimmune thyroiditis in obese chickens.肥胖鸡主要组织相容性（B）位点与自身免疫性甲状腺炎之间的关系。
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T-cell regulation of experimental autoimmune thyroiditis in the mouse.小鼠实验性自身免疫性甲状腺炎的T细胞调节
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The genetic lesions of autoimmunity.自身免疫的基因损伤。
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The genetics of thyroiditis as a prototype of human autoimmune disease.作为人类自身免疫性疾病原型的甲状腺炎遗传学
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H-2E(k) expression influences thyroiditis induction by the thyroglobulin peptide (2495-2511).H-2E(k)的表达会影响甲状腺球蛋白肽（2495-2511）诱导的甲状腺炎。
Immunogenetics. 1993;38(2):150-3. doi: 10.1007/BF00190903.
2
Genetic influences on spontaneous autoimmune thyroiditis in (CS X OS)F2 chickens.（CS×OS）F2代鸡自发性自身免疫性甲状腺炎的遗传影响
Immunogenetics. 1981;12(3-4):339-49. doi: 10.1007/BF01561675.
3
4
5
Mutations at H-2K or H-2D alter immune response phenotype of autoimmune thyroiditis.H-2K或H-2D处的突变会改变自身免疫性甲状腺炎的免疫反应表型。
Immunogenetics. 1982;15(6):625-7. doi: 10.1007/BF00347059.
6
Regulation of experimental autoimmune thyroiditis: mapping of susceptibility to the I-A subregion of the mouse H-2.实验性自身免疫性甲状腺炎的调控：小鼠H-2 I-A亚区易感性图谱
Immunogenetics. 1982;15(4):427-30. doi: 10.1007/BF00364266.
7
H-2K mutation controls immune response phenotype of autoimmune thyroiditis. Critical expression of mutant gene product in both thymus and thyroid glands.H-2K突变控制自身免疫性甲状腺炎的免疫反应表型。突变基因产物在胸腺和甲状腺中均有关键表达。
J Exp Med. 1980 Oct 1;152(4):1115-20. doi: 10.1084/jem.152.4.1115.
8
Thymic reconstitution of H-2-linked T-cell responses to thyroglobulin or insulin.H-2连锁的针对甲状腺球蛋白或胰岛素的T细胞反应的胸腺重建。
Immunogenetics. 1983;17(1):95-100. doi: 10.1007/BF00364293.
9
Dissociation and exchange of the beta 2-microglobulin subunit of HLA-A and HLA-B antigens.HLA - A和HLA - B抗原的β2 - 微球蛋白亚基的解离与交换
Proc Natl Acad Sci U S A. 1979 Nov;76(11):5834-8. doi: 10.1073/pnas.76.11.5834.
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